The function and pathogenic mechanism of filamin A.

Zhou, Jie; Kang, Xinmei; An, Hanxiang; Lv, Yun; Liu, Xin

Zhou, Jie; Kang, Xinmei; An, Hanxiang; Lv, Yun; Liu, Xin.

Afiliação

Zhou J; Department of Oncology, Xiang'an Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen 361000, Fujian, China. Electronic address: zhoujie@stu.xmu.edu.cn.
Kang X; Department of Oncology, Xiang'an Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen 361000, Fujian, China. Electronic address: kangxm28@xmu.edu.cn.
An H; Department of Oncology, Xiang'an Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen 361000, Fujian, China. Electronic address: anhanxinag@xmu.edu.cn.
Lv Y; Department of Oncology, Xiang'an Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen 361000, Fujian, China. Electronic address: lvyun@stu.xmu.edu.cn.
Liu X; Department of Oncology, Xiang'an Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen 361000, Fujian, China. Electronic address: liuxin2018@stu.xmu.edu.cn.

Gene ; 784: 145575, 2021 Jun 05.

Article em En | MEDLINE | ID: mdl-33737122

Assuntos

Resistencia a Medicamentos Antineoplásicos; Filaminas/genética; Filaminas/metabolismo; Neoplasias/genética; Citoesqueleto de Actina/metabolismo; Adesão Celular; Movimento Celular; Predisposição Genética para Doença; Humanos; Neoplasias/metabolismo

Palavras-chave

Binding protein; FLNa; Filamin A; Function; Mechanism; Tumor

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Resistencia a Medicamentos Antineoplásicos / Filaminas / Neoplasias Limite: Humans Idioma: En Revista: Gene Ano de publicação: 2021 Tipo de documento: Article

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