Your browser doesn't support javascript.
loading
GWAS of thyroid dysgenesis identifies a risk locus at 2q33.3 linked to regulation of Wnt signaling.
Narumi, Satoshi; Opitz, Robert; Nagasaki, Keisuke; Muroya, Koji; Asakura, Yumi; Adachi, Masanori; Abe, Kiyomi; Sugisawa, Chiho; Kühnen, Peter; Ishii, Tomohiro; Nöthen, Markus M; Krude, Heiko; Hasegawa, Tomonobu.
Afiliação
  • Narumi S; Department of Molecular Endocrinology, National Research Institute for Child Health and Development, Tokyo 1578535, Japan.
  • Opitz R; Department of Pediatrics, Keio University School of Medicine, Tokyo 1608582, Japan.
  • Nagasaki K; Institute for Experimental Pediatric Endocrinology, Charité - Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, 13353 Berlin, Germany.
  • Muroya K; Division of Pediatrics, Department of Homeostatic Regulation and Development, Niigata University Graduate School of Medical and Dental Sciences, Niigata 9518520, Japan.
  • Asakura Y; Department of Endocrinology and Metabolism, Kanagawa Children's Medical Center, Yokohama 2328555, Japan.
  • Adachi M; Department of Endocrinology and Metabolism, Kanagawa Children's Medical Center, Yokohama 2328555, Japan.
  • Abe K; Department of Endocrinology and Metabolism, Kanagawa Children's Medical Center, Yokohama 2328555, Japan.
  • Sugisawa C; Department of Pediatrics, Keio University School of Medicine, Tokyo 1608582, Japan.
  • Kühnen P; Department of Pediatrics, Keio University School of Medicine, Tokyo 1608582, Japan.
  • Ishii T; Institute for Experimental Pediatric Endocrinology, Charité - Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, 13353 Berlin, Germany.
  • Nöthen MM; Department of Pediatrics, Keio University School of Medicine, Tokyo 1608582, Japan.
  • Krude H; Institute of Human Genetics, University of Bonn, School of Medicine & University Hospital Bonn, 53127 Bonn, Germany.
  • Hasegawa T; Institute for Experimental Pediatric Endocrinology, Charité - Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, 13353 Berlin, Germany.
Hum Mol Genet ; 31(23): 3967-3974, 2022 11 28.
Article em En | MEDLINE | ID: mdl-35535691
Assuntos
Palavras-chave
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Disgenesia da Tireoide / Estudo de Associação Genômica Ampla Tipo de estudo: Etiology_studies / Risk_factors_studies Limite: Animals / Humans Idioma: En Revista: Hum Mol Genet Assunto da revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Japão
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Disgenesia da Tireoide / Estudo de Associação Genômica Ampla Tipo de estudo: Etiology_studies / Risk_factors_studies Limite: Animals / Humans Idioma: En Revista: Hum Mol Genet Assunto da revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Japão