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Autoimmunity-associated T cell receptors recognize HLA-B*27-bound peptides.
Yang, Xinbo; Garner, Lee I; Zvyagin, Ivan V; Paley, Michael A; Komech, Ekaterina A; Jude, Kevin M; Zhao, Xiang; Fernandes, Ricardo A; Hassman, Lynn M; Paley, Grace L; Savvides, Christina S; Brackenridge, Simon; Quastel, Max N; Chudakov, Dmitriy M; Bowness, Paul; Yokoyama, Wayne M; McMichael, Andrew J; Gillespie, Geraldine M; Garcia, K Christopher.
Afiliação
  • Yang X; Department of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, CA, USA.
  • Garner LI; Department of Structural Biology, Stanford University School of Medicine, Stanford, CA, USA.
  • Zvyagin IV; NDM Research Building, Nuffield Department of Medicine, University of Oxford, Oxford, UK.
  • Paley MA; Centre for Immuno-oncology, Nuffield Department of Medicine, University of Oxford, Oxford, UK.
  • Komech EA; Center for Precision Genome Editing and Genetic Technologies for Biomedicine, Institute of Translational Medicine, Pirogov Russian National Research Medical University, Moscow, Russian Federation.
  • Jude KM; Genomics of Adaptive Immunity Department, Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry, Moscow, Russian Federation.
  • Zhao X; Rheumatology Division, Department of Medicine, Washington University School of Medicine, St Louis, MO, USA.
  • Fernandes RA; Center for Precision Genome Editing and Genetic Technologies for Biomedicine, Institute of Translational Medicine, Pirogov Russian National Research Medical University, Moscow, Russian Federation.
  • Hassman LM; Genomics of Adaptive Immunity Department, Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry, Moscow, Russian Federation.
  • Paley GL; Department of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, CA, USA.
  • Savvides CS; Department of Structural Biology, Stanford University School of Medicine, Stanford, CA, USA.
  • Brackenridge S; Department of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, CA, USA.
  • Quastel MN; Department of Structural Biology, Stanford University School of Medicine, Stanford, CA, USA.
  • Chudakov DM; Department of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, CA, USA.
  • Bowness P; Department of Structural Biology, Stanford University School of Medicine, Stanford, CA, USA.
  • Yokoyama WM; Department of Ophthalmology, Washington University School of Medicine, St Louis, MO, USA.
  • McMichael AJ; Department of Ophthalmology, Washington University School of Medicine, St Louis, MO, USA.
  • Gillespie GM; Department of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, CA, USA.
  • Garcia KC; Department of Structural Biology, Stanford University School of Medicine, Stanford, CA, USA.
Nature ; 612(7941): 771-777, 2022 12.
Article em En | MEDLINE | ID: mdl-36477533

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Peptídeos / Receptores de Antígenos de Linfócitos T / Antígenos HLA-B / Autoimunidade Tipo de estudo: Prognostic_studies / Risk_factors_studies Limite: Humans Idioma: En Revista: Nature Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Estados Unidos

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Peptídeos / Receptores de Antígenos de Linfócitos T / Antígenos HLA-B / Autoimunidade Tipo de estudo: Prognostic_studies / Risk_factors_studies Limite: Humans Idioma: En Revista: Nature Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Estados Unidos