Post-inflammatory administration of <i>N</i>-acetylcysteine reduces inflammation and alters receptor levels in a cellular model of Parkinson's disease.

Kaya, Zeynep Bengisu; Karakoc, Elif; McLean, Pamela J; Saka, Esen; Atilla, Pergin

Post-inflammatory administration of N-acetylcysteine reduces inflammation and alters receptor levels in a cellular model of Parkinson's disease.

Kaya, Zeynep Bengisu; Karakoc, Elif; McLean, Pamela J; Saka, Esen; Atilla, Pergin.

Afiliação

Kaya ZB; Department of Neuroscience Mayo Clinic Jacksonville Florida USA.
Karakoc E; Department of Histology and Embryology Hacettepe University Faculty of Medicine Ankara Turkey.
McLean PJ; Department of Histology and Embryology Hacettepe University Faculty of Medicine Ankara Turkey.
Saka E; Department of Neuroscience Mayo Clinic Jacksonville Florida USA.
Atilla P; Department of Neurology Hacettepe University Faculty of Medicine Ankara Turkey.

FASEB Bioadv ; 5(7): 263-276, 2023 Jul.

Article em En | MEDLINE | ID: mdl-37415931

Palavras-chave

Nacetylcysteine (NAC); Parkinson's disease; alphasynuclein (αsyn); apoptosis; neuroinflammation

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Prognostic_studies / Risk_factors_studies Idioma: En Revista: FASEB Bioadv Ano de publicação: 2023 Tipo de documento: Article

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