Disruption of extracellular redox balance drives persistent lung fibrosis and impairs fibrosis resolution.

Cui, Ye; Yang, Zeran; Lv, Zhe; Lei, Jianfeng

Cui, Ye; Yang, Zeran; Lv, Zhe; Lei, Jianfeng.

Afiliação

Cui Y; Department of Immunology, School of Basic Medical Sciences, Capital Medical University, Beijing 100069, People's Republic of China. Electronic address: yecui@outlook.com.
Yang Z; Interventional Radiology, Beijing Friendship Hospital, Capital Medical University, Beijing 100050, People's Republic of China.
Lv Z; Department of Immunology, School of Basic Medical Sciences, Capital Medical University, Beijing 100069, People's Republic of China.
Lei J; Medical Imaging Laboratory, Research Core Facilities, Capital Medical University, Beijing 100069, People's Republic of China.

Biochim Biophys Acta Mol Basis Dis ; 1869(8): 166842, 2023 12.

Article em En | MEDLINE | ID: mdl-37558008

Assuntos

Fibrose Pulmonar; Camundongos; Animais; Fibrose Pulmonar/induzido quimicamente; Fibrose Pulmonar/genética; Fibrose Pulmonar/metabolismo; Espécies Reativas de Oxigênio/metabolismo; Fibrose; Bleomicina; Oxirredução

Palavras-chave

Apoptosis resistance; Extracellular superoxide dismutase; Fibroblasts; Lung fibrosis; Reactive oxygen species; Resolution

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Fibrose Pulmonar Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Revista: Biochim Biophys Acta Mol Basis Dis Ano de publicação: 2023 Tipo de documento: Article

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