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ß-hydroxybutyrate administered at reperfusion reduces infarct size and preserves cardiac function by improving mitochondrial function through autophagy in male mice.
Chu, Yuxin; Hua, Yutao; He, Lihao; He, Jin; Chen, Yunxi; Yang, Jing; Mahmoud, Ismail; Zeng, Fanfang; Zeng, Xiaochang; Benavides, Gloria A; Darley-Usmar, Victor M; Young, Martin E; Ballinger, Scott W; Prabhu, Sumanth D; Zhang, Cheng; Xie, Min.
Afiliação
  • Chu Y; National Key Laboratory for Innovation and Transformation of Luobing Theory, The Key Laboratory of Cardiovascular Remodeling and Function Research, Chinese Ministry of Education, Chinese National Health Commission and Chinese Academy of Medical Sciences, Department of Cardiology, Qilu Hospital of Sh
  • Hua Y; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • He L; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • He J; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • Chen Y; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • Yang J; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • Mahmoud I; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • Zeng F; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA; Department of Cardiovascular Disease, Fuwai Hospital Chinese Academy of Medical Sciences Shenzhen, Shenzhen 518020, China.
  • Zeng X; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA; Department of Cardiovascular Disease, Fuwai Hospital Chinese Academy of Medical Sciences Shenzhen, Shenzhen 518020, China.
  • Benavides GA; Department of Pathology, Division of Molecular and Cellular Pathology, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • Darley-Usmar VM; Department of Pathology, Division of Molecular and Cellular Pathology, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • Young ME; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • Ballinger SW; Department of Pathology, Division of Molecular and Cellular Pathology, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • Prabhu SD; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA.
  • Zhang C; National Key Laboratory for Innovation and Transformation of Luobing Theory, The Key Laboratory of Cardiovascular Remodeling and Function Research, Chinese Ministry of Education, Chinese National Health Commission and Chinese Academy of Medical Sciences, Department of Cardiology, Qilu Hospital of Sh
  • Xie M; Department of Medicine, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35233, USA. Electronic address: mxie@uabmc.edu.
J Mol Cell Cardiol ; 186: 31-44, 2024 01.
Article em En | MEDLINE | ID: mdl-37979443
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Traumatismo por Reperfusão Miocárdica / Infarto do Miocárdio com Supradesnível do Segmento ST / Insuficiência Cardíaca Limite: Animals / Humans / Male Idioma: En Revista: J Mol Cell Cardiol Ano de publicação: 2024 Tipo de documento: Article
Texto completo
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XML
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Traumatismo por Reperfusão Miocárdica / Infarto do Miocárdio com Supradesnível do Segmento ST / Insuficiência Cardíaca Limite: Animals / Humans / Male Idioma: En Revista: J Mol Cell Cardiol Ano de publicação: 2024 Tipo de documento: Article