STAT3 as a mediator of BCR-ABL1-independent resistance in chronic myeloid leukemia.

Eiring, A M; Kraft, I L; Page, B D; O'Hare, T; Gunning, P T; Deininger, M W

Eiring, A M; Kraft, I L; Page, B D; O'Hare, T; Gunning, P T; Deininger, M W.

Afiliação

Eiring AM; Division of Hematology and Hematologic Malignancies, University of Utah, Huntsman Cancer Institute , Salt Lake City, UT, USA.
Kraft IL; Division of Hematology and Hematologic Malignancies, University of Utah, Huntsman Cancer Institute , Salt Lake City, UT, USA.
Page BD; Department of Chemical and Physical Sciences, University of Toronto , Toronto, ON, Canada.
O'Hare T; Division of Hematology and Hematologic Malignancies, University of Utah, Huntsman Cancer Institute , Salt Lake City, UT, USA.
Gunning PT; Department of Chemical and Physical Sciences, University of Toronto , Toronto, ON, Canada.
Deininger MW; Division of Hematology and Hematologic Malignancies, University of Utah, Huntsman Cancer Institute , Salt Lake City, UT, USA.

Leuk Suppl ; 3(Suppl 1): S5-6, 2014 Dec.

Article em En | MEDLINE | ID: mdl-27175272

Palavras-chave

BCR-ABL1-dependent resistance; BCR-ABL1-independent resistance; BP-5-087; CML stem cell; STAT3; TKI resistance; pSTAT3Y705

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Revista: Leuk Suppl Ano de publicação: 2014 Tipo de documento: Article País de afiliação: Estados Unidos

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