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Toll-like receptor 9 signaling promotes autophagy and apoptosis via divergent functions of the p38/JNK pathway in human salivary gland cells.
Fu, Jiayao; Shi, Huan; Cao, Ningning; Wu, Shufeng; Zhan, Tianle; Xie, Lisong; Wang, Zhijun; Ye, Lei; Li, Changyi; Shen, Yichao; Yu, Chuangqi; Zheng, Lingyan.
Afiliação
  • Fu J; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China.
  • Shi H; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China.
  • Cao N; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China.
  • Wu S; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China.
  • Zhan T; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China.
  • Xie L; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China.
  • Wang Z; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China.
  • Ye L; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China.
  • Li C; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China.
  • Shen Y; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China.
  • Yu C; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China. Ele
  • Zheng L; Department of Oral Surgery, Shanghai Ninth People's Hospital, College of Stomatology, Shanghai Jiao Tong University School of Medicine, China; National Clinical Research Center of Oral Disease, China; Shanghai Key Laboratory of Stomatology & Shanghai Research Institute of Stomatology, China. Ele
Exp Cell Res ; 375(2): 51-59, 2019 02 15.
Article em En | MEDLINE | ID: mdl-30610847
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Glândulas Salivares / Autofagia / Apoptose / Sistema de Sinalização das MAP Quinases / Receptor Toll-Like 9 Limite: Humans Idioma: En Revista: Exp Cell Res Ano de publicação: 2019 Tipo de documento: Article País de afiliação: China
Texto completo
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Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Glândulas Salivares / Autofagia / Apoptose / Sistema de Sinalização das MAP Quinases / Receptor Toll-Like 9 Limite: Humans Idioma: En Revista: Exp Cell Res Ano de publicação: 2019 Tipo de documento: Article País de afiliação: China