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GCH1 induces immunosuppression through metabolic reprogramming and IDO1 upregulation in triple-negative breast cancer.
Wei, Jin-Li; Wu, Si-Yu; Yang, Yun-Song; Xiao, Yi; Jin, Xi; Xu, Xiao-En; Hu, Xin; Li, Da-Qiang; Jiang, Yi-Zhou; Shao, Zhi-Ming.
Afiliação
  • Wei JL; Department of Breast Surgery, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Wu SY; Department of Breast Surgery, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Yang YS; Department of Breast Surgery, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Xiao Y; Department of Breast Surgery, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Jin X; Department of Breast Surgery, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Xu XE; Cancer Institute, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Hu X; Cancer Institute, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Li DQ; Cancer Institute, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Jiang YZ; Department of Breast Surgery, Fudan University Shanghai Cancer Center, Shanghai, China zhimingshao@yahoo.com yizhoujiang@fudan.edu.cn.
  • Shao ZM; Department of Breast Surgery, Fudan University Shanghai Cancer Center, Shanghai, China zhimingshao@yahoo.com yizhoujiang@fudan.edu.cn.
J Immunother Cancer ; 9(7)2021 07.
Article em En | MEDLINE | ID: mdl-34281987
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Linfócitos T Reguladores / Indolamina-Pirrol 2,3,-Dioxigenase / Neoplasias de Mama Triplo Negativas / GTP Cicloidrolase Limite: Animals / Female / Humans Idioma: En Revista: J Immunother Cancer Ano de publicação: 2021 Tipo de documento: Article País de afiliação: China
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Linfócitos T Reguladores / Indolamina-Pirrol 2,3,-Dioxigenase / Neoplasias de Mama Triplo Negativas / GTP Cicloidrolase Limite: Animals / Female / Humans Idioma: En Revista: J Immunother Cancer Ano de publicação: 2021 Tipo de documento: Article País de afiliação: China