Dissociation of SH3 and cysteine-rich domain 3 and junctophilin 1 from dihydropyridine receptor in dystrophin-deficient muscles.

Ashida, Yuki; Himori, Koichi; Tokuda, Nao; Naito, Azuma; Yamauchi, Nao; Takenaka-Ninagawa, Nana; Aoki, Yoshitsugu; Sakurai, Hidetoshi; Yamada, Takashi

Ashida, Yuki; Himori, Koichi; Tokuda, Nao; Naito, Azuma; Yamauchi, Nao; Takenaka-Ninagawa, Nana; Aoki, Yoshitsugu; Sakurai, Hidetoshi; Yamada, Takashi.

Afiliação

Ashida Y; Graduate School of Health Sciences, Sapporo Medical University, Sapporo, Japan.
Himori K; Japan Society for the Promotion of Science, Tokyo, Japan.
Tokuda N; Graduate School of Health Sciences, Sapporo Medical University, Sapporo, Japan.
Naito A; Japan Society for the Promotion of Science, Tokyo, Japan.
Yamauchi N; Graduate School of Health Sciences, Sapporo Medical University, Sapporo, Japan.
Takenaka-Ninagawa N; Graduate School of Health Sciences, Sapporo Medical University, Sapporo, Japan.
Aoki Y; Graduate School of Health Sciences, Sapporo Medical University, Sapporo, Japan.
Sakurai H; Center for iPS Cell Research and Application (CiRA), Kyoto University, Kyoto, Japan.
Yamada T; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Tokyo, Japan.

Am J Physiol Cell Physiol ; 323(3): C885-C895, 2022 09 01.

Article em En | MEDLINE | ID: mdl-35912995

Assuntos

Canais de Cálcio Tipo L; Distrofia Muscular de Duchenne; Proteínas Adaptadoras de Transdução de Sinal/metabolismo; Animais; Cálcio/metabolismo; Canais de Cálcio Tipo L/metabolismo; Calpaína/metabolismo; Cisteína/metabolismo; Distrofina/genética; Distrofina/metabolismo; Proteínas de Membrana; Camundongos; Camundongos Endogâmicos mdx; Debilidade Muscular/metabolismo; Músculo Esquelético/metabolismo; Distrofia Muscular de Duchenne/genética; Distrofia Muscular de Duchenne/metabolismo

Palavras-chave

STAC3; calpain; eccentric contraction; junctophilin 1; muscular dystrophy

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Distrofia Muscular de Duchenne / Canais de Cálcio Tipo L Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Revista: Am J Physiol Cell Physiol Assunto da revista: FISIOLOGIA Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Japão

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