Game performance and intermittent hypoxic training.

Live high-train low altitude exposure simulated by hypoxic devices may improve athletic performance. In this study, intermittent normobaric hypoxia was achieved with the GO2altitude hypoxicator to determine its effects on sea level performance in rugby players. Ten players were randomly assigned to two groups. Players in each group received 14 sessions of either hypoxic (10-15% O(2)) or normoxic (21% O(2)) exposure at rest over 14 consecutive days in a single blind fashion. Various performance measures were obtained consecutively in a single testing session pre- and post-exposure. Effects of hypoxic exposure on maximum speed and sprint times were trivial (<1.0%) but unclear (90% likely range, +/-5% to +/-9%). In rugby simulation, hypoxic exposure produced impairments of peak power in two scrums (15%, +/-8%; 9%, +/-7%) and impairments of time in offensive sprints (7%, +/-8%) and tackle sprints (11%, +/-9%). Pending further research, rugby players would be unwise to use normobaric intermittent hypoxic exposure to prepare for games at sea level.

The effect of intermittent hypoxic training via a hypoxic inhaler on physiological and performance measures in rowers: a pilot study.

Intermittent hypoxic training and discontinuous exposure to altitude were used to improve performance at sea level in elite rowers. Altitude was simulated with a newly patented device which allowed athletes to experience altitude by re-breathing oxygen-depleted expired air. Seven elite rowers (five females, two males) used inhalers for a 90-min supervised daily session (alternating 6 min on and 4 min off) for 3 weeks, while four female elite rowers used placebo devices in the same sessions. The inhalers were adjusted to produce a progressive decrease in oxygen saturation over the 3 weeks (initially 90%; finally 80%). Immediately before and 7-10 days after altitude exposure, the rowers performed an incremental lactate test to determine power output equivalent to 4 mM [BLa], a 500-m time trial and a 5000-m time trial, all on a rowing ergometer. Relative to the control group, the altitude group showed a slight improvement in mean power for the 5000-m time trial (0.6%, 90% likely limits +/-3.7%), and a substantial impairment in mean power for the 500-m trial (2.2%, +/-4.1%). Power at 4-mM lactate declined in both groups, but overall the altitude group improved by 0.4% (+/-3.5%) relative to control. The device represents a practical way to simulate altitude exposure, but it is unlikely to have major effects on performance of elite rowers.

Sea-level performance in runners using altitude tents: a field study.

In this study of effects of simulated altitude exposure on sea-level performance, 10 competitive runners slept in a hypoxic environment achieved with tents for 9.8+/-1.3 h.d(-1) (mean+/-standard deviation) for 24 days-30 days at 2500-3500 m (PIO2=117-103 mmHg) above sea level. The altitude group and a control group of 10 runners performed usual training (PIO2=149 mmHg). At approximately 4-wk intervals before and after exposure both groups performed an incremental test for lactate threshold. The altitude group performed an additional test, a treadmill run to exhaustion lasting approximately 5 min. One week following exposure lactate threshold speed of the altitude group relative to the control group increased by 1.2% (90% likely limits +/-3.1%), but the effect became slightly negative after controlling for baseline differences in running speed between the groups. A 16% increase in time to exhaustion was observed in the altitude group, equivalent to a 1.9% (+/-1.4%) increase in speed in a time trial. Change in performance had an unclear relationship to total altitude exposure, genotype for angiotensin converting enzyme, and change in haemoglobin concentration. Our findings are consistent with little or no effect of use of altitude tents on sea-level performance.