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1.
Skatole-induced p38 and JNK activation coordinately upregulates, whereas AhR activation partially attenuates TNFα expression in intestinal epithelial cells.
Biosci Biotechnol Biochem;
87(6): 611-619, 2023 May 19.
Artículo
en Inglés
| MEDLINE | ID: mdl-36941128
2.
Activation of the TLR4-JNK but not the TLR4-ERK pathway induced by indole-3-acetic acid exerts anti-proliferative effects on Caco-2 cells.
Biosci Biotechnol Biochem;
87(8): 839-849, 2023 Jul 24.
Artículo
en Inglés
| MEDLINE | ID: mdl-37147026
3.
Suppression of TNFα expression induced by indole-3-acetic acid is not mediated by AhR activation in Caco-2 cells.
Biosci Biotechnol Biochem;
85(4): 902-906, 2021 Mar 24.
Artículo
en Inglés
| MEDLINE | ID: mdl-33580679
4.
TLR4 may be a novel indole-3-acetic acid receptor that is implicated in the regulation of CYP1A1 and TNFα expression depending on the culture stage of Caco-2 cells.
Biosci Biotechnol Biochem;
85(9): 2011-2021, 2021 Aug 25.
Artículo
en Inglés
| MEDLINE | ID: mdl-34245564
5.
Microcystin-LR incorporated into colonic cells through probenecid-sensitive transporters leads to upregulated MCP-1 expression induced by JNK activation.
Toxicol Rep;
9: 937-944, 2022.
Artículo
en Inglés
| MEDLINE | ID: mdl-35875256
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