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1.
Blocking the CTLA-4 and PD-1 pathways during pulmonary paracoccidioidomycosis improves immunity, reduces disease severity, and increases the survival of infected mice.
Front Immunol;
15: 1347318, 2024.
Artículo
en Inglés
| MEDLINE | ID: mdl-38500881
2.
Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy.
Front Immunol;
14: 1039244, 2023.
Artículo
en Inglés
| MEDLINE | ID: mdl-36776848
3.
The immunosuppressive activity of myeloid-derived suppressor cells in murine Paracoccidioidomycosis relies on Indoleamine 2,3-dioxygenase activity and Dectin-1 and TLRs signaling.
Sci Rep;
13(1): 12391, 2023 07 31.
Artículo
en Inglés
| MEDLINE | ID: mdl-37524886
4.
Transcriptional profiling of a fungal granuloma reveals a low metabolic activity of Paracoccidioides brasiliensis yeasts and an actively regulated host immune response.
Front Cell Infect Microbiol;
13: 1268959, 2023.
Artículo
en Inglés
| MEDLINE | ID: mdl-37868350
5.
Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses.
Sci Rep;
10(1): 11312, 2020 07 09.
Artículo
en Inglés
| MEDLINE | ID: mdl-32647342
6.
The Syk-Coupled C-Type Lectin Receptors Dectin-2 and Dectin-3 Are Involved in Paracoccidioides brasiliensis Recognition by Human Plasmacytoid Dendritic Cells.
Front Immunol;
9: 464, 2018.
Artículo
en Inglés
| MEDLINE | ID: mdl-29616019
7.
The IDO-AhR Axis Controls Th17/Treg Immunity in a Pulmonary Model of Fungal Infection.
Front Immunol;
8: 880, 2017.
Artículo
en Inglés
| MEDLINE | ID: mdl-28791025
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