Procyanidin dimer B2-mediated IRAK-M induction negatively regulates TLR4 signaling in macrophages.
Biochem Biophys Res Commun
; 438(1): 122-8, 2013 Aug 16.
Article
en En
| MEDLINE
| ID: mdl-23872113
Palabras clave
Cytokine; DTT; FITC; IFN; IL; IL-1 receptor-associated kinase; IRAK; Iκ-B; LPS; MAPK; Mitogen-activated protein kinases; MyD88; NF-κB; Nuclear factor κB; PBS; PE; PMSF; Procyanidin B2; SDS; SOCS1; TIR; TIR domain-containing adapter-inducing interferon-ß; TLR; TNF-α; TOLLIP; TRIF; Toll-like receptor; dithiothreitol; fluorescein isothiocyanate; inhibitor of κB; interferon; interleukin; lipopolysaccharide; mitogen-activated protein kinase; myeloid differentiation factor 88; nuclear factor-κB; phenylmethanesulfonyl fluoride; phosphate-buffered saline; phycoerythrin; sodium dodecyl sulfate; suppressor of cytokine signaling 1; toll-interacting protein; toll-like receptor; toll/interleukin-1 receptor homology; tumor necrosis factor-α
Texto completo:
1
Bases de datos:
MEDLINE
Asunto principal:
Catequina
/
Biflavonoides
/
Proantocianidinas
/
Receptor Toll-Like 4
/
Quinasas Asociadas a Receptores de Interleucina-1
/
Macrófagos
Límite:
Animals
Idioma:
En
Revista:
Biochem Biophys Res Commun
Año:
2013
Tipo del documento:
Article
País de afiliación:
Corea del Sur