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RelA/p65 inhibition prevents tendon adhesion by modulating inflammation, cell proliferation, and apoptosis.
Chen, Shuai; Jiang, Shichao; Zheng, Wei; Tu, Bing; Liu, Shen; Ruan, Hongjiang; Fan, Cunyi.
Afiliación
  • Chen S; Department of Orthopaedics, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, 600 Yishan Road, Shanghai 200233, People's Republic of China.
  • Jiang S; Department of Orthopaedics, Shandong Provincial Hospital Affiliated to Shandong University, No. 324 Jingwu Road, Jinan 250021, Shandong, People's Republic of China.
  • Zheng W; Department of Orthopaedics, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, 600 Yishan Road, Shanghai 200233, People's Republic of China.
  • Tu B; Department of Orthopaedics, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, 600 Yishan Road, Shanghai 200233, People's Republic of China.
  • Liu S; Department of Orthopaedics, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, 600 Yishan Road, Shanghai 200233, People's Republic of China.
  • Ruan H; Department of Orthopaedics, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, 600 Yishan Road, Shanghai 200233, People's Republic of China.
  • Fan C; Department of Orthopaedics, Shanghai Jiao Tong University Affiliated Sixth People's Hospital, 600 Yishan Road, Shanghai 200233, People's Republic of China.
Cell Death Dis ; 8(3): e2710, 2017 03 30.
Article en En | MEDLINE | ID: mdl-28358376

Texto completo: 1 Bases de datos: MEDLINE Asunto principal: Tendones / Adherencias Tisulares / Apoptosis / Proliferación Celular / Factor de Transcripción ReIA Tipo de estudio: Prognostic_studies Límite: Adult / Female / Humans / Male / Middle aged Idioma: En Revista: Cell Death Dis Año: 2017 Tipo del documento: Article

Texto completo: 1 Bases de datos: MEDLINE Asunto principal: Tendones / Adherencias Tisulares / Apoptosis / Proliferación Celular / Factor de Transcripción ReIA Tipo de estudio: Prognostic_studies Límite: Adult / Female / Humans / Male / Middle aged Idioma: En Revista: Cell Death Dis Año: 2017 Tipo del documento: Article