Targeted deletion of Fgf9 in tendon disrupts mineralization of the developing enthesis.

Ganji, Elahe; Leek, Connor; Duncan, William; Patra, Debabrata; Ornitz, David M; Killian, Megan L

Ganji, Elahe; Leek, Connor; Duncan, William; Patra, Debabrata; Ornitz, David M; Killian, Megan L.

Afiliación

Ganji E; Department of Orthopaedic Surgery, Michigan Medicine, Michigan, Ann Arbor, USA.
Leek C; Department of Mechanical Engineering, University of Delaware, Delaware, Newark, USA.
Duncan W; Beckman Institute for Advanced Science and Technology, University of Illinois at Urbana-Champaign, 61801, IL, Urbana, United States.
Patra D; Department of Biomedical Engineering, University of Delaware, Delaware, Newark, USA.
Ornitz DM; Department of Orthopaedic Surgery, Michigan Medicine, Michigan, Ann Arbor, USA.
Killian ML; Department of Biomedical Engineering, University of Delaware, Delaware, Newark, USA.

FASEB J ; 37(3): e22777, 2023 03.

Article en En | MEDLINE | ID: mdl-36734881

Asunto(s)

Factor 9 de Crecimiento de Fibroblastos; Tendones; Ratones; Animales; Factor 9 de Crecimiento de Fibroblastos/genética; Factor 9 de Crecimiento de Fibroblastos/metabolismo; Tendones/metabolismo; Huesos; Desarrollo Óseo/genética; Condrogénesis

Palabras clave

attachment; enthesis; fibroblast growth factor; musculoskeletal; postnatal; tendon

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Texto completo: 1 Bases de datos: MEDLINE Asunto principal: Tendones / Factor 9 de Crecimiento de Fibroblastos Límite: Animals Idioma: En Revista: FASEB J Asunto de la revista: BIOLOGIA / FISIOLOGIA Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos

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