Detalhe da pesquisa
1.
Increased Atrial ß-Adrenergic Receptors and GRK-2 Gene Expression Can Play a Fundamental Role in Heart Failure After Repair of Congenital Heart Disease with Cardiopulmonary Bypass.
Pediatr Cardiol
; 38(4): 734-745, 2017 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-28214967
2.
Early detection of doxorubicin myocardial injury by ultrasonic tissue characterization in an experimental animal model.
Cardiovasc Ultrasound
; 10: 40, 2012 Oct 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-23046747
3.
Disruption of sarcolemmal dystrophin and beta-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis.
Lab Invest
; 90(4): 531-42, 2010 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-20142806
4.
Isoproterenol induces primary loss of dystrophin in rat hearts: correlation with myocardial injury.
Int J Exp Pathol
; 89(5): 367-81, 2008 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-18808529
5.
Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis.
PLoS One
; 8(7): e68809, 2013.
Artigo
em Inglês
| MEDLINE | ID: mdl-23935889
6.
Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy.
Microbes Infect
; 14(1): 59-68, 2012 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-21914488
7.
Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy.
Eur J Pharmacol
; 670(2-3): 541-53, 2011 Nov 30.
Artigo
em Inglês
| MEDLINE | ID: mdl-21946105
8.
Coronary microvascular disease in chronic Chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms.
PLoS Negl Trop Dis
; 4(8)2010 Aug 31.
Artigo
em Inglês
| MEDLINE | ID: mdl-20824217
9.
Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins.
Shock
; 33(3): 322-31, 2010 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-20160610