Detalhe da pesquisa
1.
Slow Ca²âº sparks de-synchronize Ca²âº release in failing cardiomyocytes: evidence for altered configuration of Ca²âº release units?
J Mol Cell Cardiol
; 58: 41-52, 2013 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-23376034
2.
Control of Ca2+ release by action potential configuration in normal and failing murine cardiomyocytes.
Biophys J
; 99(5): 1377-86, 2010 Sep 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-20816049
3.
Sodium accumulation promotes diastolic dysfunction in end-stage heart failure following Serca2 knockout.
J Physiol
; 588(Pt 3): 465-78, 2010 Feb 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-20008467
4.
Tamoxifen administration routes and dosage for inducible Cre-mediated gene disruption in mouse hearts.
Transgenic Res
; 19(4): 715-25, 2010 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-19894134
5.
Slowing of cardiomyocyte Ca2+ release and contraction during heart failure progression in postinfarction mice.
Am J Physiol Heart Circ Physiol
; 296(4): H1069-79, 2009 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-19201998
6.
Increased cardiomyocyte function and Ca2+ transients in mice during early congestive heart failure.
J Mol Cell Cardiol
; 43(2): 177-86, 2007 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-17574269
7.
Contribution of the Na+/Ca2+ exchanger to rapid Ca2+ release in cardiomyocytes.
Biophys J
; 91(3): 779-92, 2006 Aug 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-16679359
8.
T-tubule disorganization and reduced synchrony of Ca2+ release in murine cardiomyocytes following myocardial infarction.
J Physiol
; 574(Pt 2): 519-33, 2006 Jul 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-16709642