Detalhe da pesquisa
1.
Alternative splicing redefines landscape of commonly mutated genes in acute myeloid leukemia.
Proc Natl Acad Sci U S A
; 118(15)2021 04 13.
Artigo
em Inglês
| MEDLINE | ID: mdl-33876749
2.
Widespread JNK-dependent alternative splicing induces a positive feedback loop through CELF2-mediated regulation of MKK7 during T-cell activation.
Genes Dev
; 29(19): 2054-66, 2015 Oct 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-26443849
3.
Reciprocal regulation of hnRNP C and CELF2 through translation and transcription tunes splicing activity in T cells.
Nucleic Acids Res
; 48(10): 5710-5719, 2020 06 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-32338744
4.
Ancient antagonism between CELF and RBFOX families tunes mRNA splicing outcomes.
Genome Res
; 27(8): 1360-1370, 2017 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-28512194
5.
Phosphoproteomics reveals that glycogen synthase kinase-3 phosphorylates multiple splicing factors and is associated with alternative splicing.
J Biol Chem
; 292(44): 18240-18255, 2017 11 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-28916722
6.
Induced transcription and stability of CELF2 mRNA drives widespread alternative splicing during T-cell signaling.
Proc Natl Acad Sci U S A
; 112(17): E2139-48, 2015 Apr 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-25870297
7.
Global analysis of physical and functional RNA targets of hnRNP L reveals distinct sequence and epigenetic features of repressed and enhanced exons.
RNA
; 21(12): 2053-66, 2015 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-26437669
8.
TRAP150 interacts with the RNA-binding domain of PSF and antagonizes splicing of numerous PSF-target genes in T cells.
Nucleic Acids Res
; 43(18): 9006-16, 2015 Oct 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-26261210
9.
An optogenetic gene expression system with rapid activation and deactivation kinetics.
Nat Chem Biol
; 10(3): 196-202, 2014 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-24413462
10.
Position-dependent activity of CELF2 in the regulation of splicing and implications for signal-responsive regulation in T cells.
RNA Biol
; 13(6): 569-81, 2016 06 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-27096301
11.
Oxidative-stress-induced nuclear to cytoplasmic relocalization is required for Not4-dependent cyclin C destruction.
J Cell Sci
; 125(Pt 4): 1015-26, 2012 Feb 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-22421358
12.
Alternative splicing of HDAC7 regulates its interaction with 14-3-3 proteins to alter histone marks and target gene expression.
Cell Rep
; 42(3): 112273, 2023 03 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-36933216
13.
A disease-associated polymorphism alters splicing of the human CD45 phosphatase gene by disrupting combinatorial repression by heterogeneous nuclear ribonucleoproteins (hnRNPs).
J Biol Chem
; 286(22): 20043-53, 2011 Jun 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-21507955
14.
Ume6p is required for germination and early colony development of yeast ascospores.
FEMS Yeast Res
; 11(1): 104-13, 2011 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-21059190
15.
The Sin3p PAH domains provide separate functions repressing meiotic gene transcription in Saccharomyces cerevisiae.
Eukaryot Cell
; 9(12): 1835-44, 2010 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-20971827
16.
Pds1p is required for meiotic recombination and prophase I progression in Saccharomyces cerevisiae.
Genetics
; 181(1): 65-79, 2009 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-19001291
17.
Viral-induced alternative splicing of host genes promotes influenza replication.
Elife
; 92020 12 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-33269701
18.
RNA Binding Protein CELF2 Regulates Signal-Induced Alternative Polyadenylation by Competing with Enhancers of the Polyadenylation Machinery.
Cell Rep
; 28(11): 2795-2806.e3, 2019 09 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-31509743
19.
Co-regulatory activity of hnRNP K and NS1-BP in influenza and human mRNA splicing.
Nat Commun
; 9(1): 2407, 2018 06 19.
Artigo
em Inglês
| MEDLINE | ID: mdl-29921878
20.
Regulation of the oxidative stress response through Slt2p-dependent destruction of cyclin C in Saccharomyces cerevisiae.
Genetics
; 172(3): 1477-86, 2006 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-16387872