Detalhe da pesquisa
1.
Cumulative haploinsufficiency and triplosensitivity drive aneuploidy patterns and shape the cancer genome.
Cell
; 155(4): 948-62, 2013 Nov 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-24183448
2.
Genetic Screens Reveal FEN1 and APEX2 as BRCA2 Synthetic Lethal Targets.
Mol Cell
; 73(5): 885-899.e6, 2019 03 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-30686591
3.
Genetic analysis of cancer drivers reveals cohesin and CTCF as suppressors of PD-L1.
Proc Natl Acad Sci U S A
; 119(7)2022 02 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-35149558
4.
Functional genomics reveals that tumors with activating phosphoinositide 3-kinase mutations are dependent on accelerated protein turnover.
Genes Dev
; 30(24): 2684-2695, 2016 12 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-28087713
5.
LPS ligand and culture additives improve production of monomeric MD-1 and 2 in Pichia pastoris by decreasing aggregation and intermolecular disulfide bonding.
Protein Expr Purif
; 76(2): 173-83, 2011 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-21130168
6.
The T790M mutation in EGFR kinase causes drug resistance by increasing the affinity for ATP.
Proc Natl Acad Sci U S A
; 105(6): 2070-5, 2008 Feb 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-18227510
7.
Identification of FUBP1 as a Long Tail Cancer Driver and Widespread Regulator of Tumor Suppressor and Oncogene Alternative Splicing.
Cell Rep
; 28(13): 3435-3449.e5, 2019 09 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-31553912
8.
Hirudin binding reveals key determinants of thrombin allostery.
J Biol Chem
; 280(29): 26997-7003, 2005 Jul 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-15923186