Detalhe da pesquisa
1.
dATP elevation induces myocardial metabolic remodeling to support improved cardiac function.
J Mol Cell Cardiol
; 175: 1-12, 2023 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-36470336
2.
Cardiac myosin activation with 2-deoxy-ATP via increased electrostatic interactions with actin.
Proc Natl Acad Sci U S A
; 116(23): 11502-11507, 2019 06 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-31110001
3.
Impact of etiology on force and kinetics of left ventricular end-stage failing human myocardium.
J Mol Cell Cardiol
; 156: 7-19, 2021 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-33766524
4.
Etiology-dependent impairment of relaxation kinetics in right ventricular end-stage failing human myocardium.
J Mol Cell Cardiol
; 121: 81-93, 2018 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-29981798
5.
Low levels of Survival Motor Neuron protein are sufficient for normal muscle function in the SMNΔ7 mouse model of SMA.
Hum Mol Genet
; 24(21): 6160-73, 2015 Nov 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-26276812
6.
Early treatment with lisinopril and spironolactone preserves cardiac and skeletal muscle in Duchenne muscular dystrophy mice.
Circulation
; 124(5): 582-8, 2011 Aug 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-21768542
7.
Prednisolone attenuates improvement of cardiac and skeletal contractile function and histopathology by lisinopril and spironolactone in the mdx mouse model of Duchenne muscular dystrophy.
PLoS One
; 9(2): e88360, 2014.
Artigo
em Inglês
| MEDLINE | ID: mdl-24551095
8.
The force-temperature relationship in healthy and dystrophic mouse diaphragm; implications for translational study design.
Front Physiol
; 3: 422, 2012.
Artigo
em Inglês
| MEDLINE | ID: mdl-23162469