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Clin Transplant ; 25(4): E381-9, 2011.
Artigo em Inglês | MEDLINE | ID: mdl-21418327

RESUMO

BACKGROUND: Bronchiolitis obliterans syndrome (BOS) is the leading cause of morbidity/mortality in lung-transplant recipients (LTRs). Recent studies demonstrated that azithromycin (AZI) can improve graft function in BOS. We here investigated whether a 12-month course of AZI could more efficiently impact the course of BOS if administered early in BOS development. METHODS: Using a retrospective study, we examined AZI effects on graft function in 62 LTRs: 25 with potential BOS (BOS 0-p) and 37 with BOS grade 1-3. Response was defined as a ≥ 10% FEV(1) increase. Bronchoalveolar (BAL) neutrophilia and levels of IL-8, 8-isoprostane and other plasma cytokines were analyzed as parameters of lung or systemic inflammation. RESULTS: After 12-month AZI, 13 patients were responders, 35 had graft function stabilization, and 14 further deteriorated. The frequency of responders was significantly higher in LTRs with BOS 0-p (44%) than in those with BOS grade 1-3 (6%). No association was found between BAL features and AZI response while a significant decrease in plasma levels of IL-8, MCP-1, I-309, MIP-1α, and TNF-α was detected. CONCLUSIONS: Long-term AZI can improve or stabilize lung graft function in LTRs with BOS, but the treatment impacts the course of the disease more efficiently if administered in BOS 0-p.


Assuntos
Azitromicina/uso terapêutico , Bronquiolite Obliterante/terapia , Rejeição de Enxerto/tratamento farmacológico , Transplante de Pulmão/efeitos adversos , Pneumonia/tratamento farmacológico , Antibacterianos/uso terapêutico , Bronquiolite Obliterante/complicações , Quimiocina CCL3/metabolismo , Doença Crônica , Feminino , Citometria de Fluxo , Seguimentos , Rejeição de Enxerto/etiologia , Humanos , Interleucina-8/metabolismo , Masculino , Pessoa de Meia-Idade , Pneumonia/etiologia , Prognóstico , Testes de Função Respiratória , Estudos Retrospectivos , Taxa de Sobrevida , Transplante Homólogo , Fator de Necrose Tumoral alfa/metabolismo
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