Detalhe da pesquisa
1.
Innate phase production of IFN-γ by memory and effector T cells expressing early activation marker CD69 during infection with Cryptococcus deneoformans in the lungs.
Infect Immun
; : e0002424, 2024 May 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-38700335
2.
Production of IL-17A at Innate Immune Phase Leads to Decreased Th1 Immune Response and Attenuated Host Defense against Infection with Cryptococcus deneoformans.
J Immunol
; 205(3): 686-698, 2020 08 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-32561568
3.
Dectin-2 deficiency promotes Th2 response and mucin production in the lungs after pulmonary infection with Cryptococcus neoformans.
Infect Immun
; 83(2): 671-81, 2015 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-25422263
4.
Defect of CARD9 leads to impaired accumulation of gamma interferon-producing memory phenotype T cells in lungs and increased susceptibility to pulmonary infection with Cryptococcus neoformans.
Infect Immun
; 82(4): 1606-15, 2014 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-24470469
5.
Cryptococcus neoformans Infection in Mice Lacking Type I Interferon Signaling Leads to Increased Fungal Clearance and IL-4-Dependent Mucin Production in the Lungs.
PLoS One
; 10(9): e0138291, 2015.
Artigo
em Inglês
| MEDLINE | ID: mdl-26384031
6.
Generation of Mouse STO Feeder Cell Lines That Confer Resistance to Several Types of Selective Drugs.
Cell Med
; 3(1-3): 97-102, 2012 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-28058186