RESUMO
A 63-year-old woman with atrial fibrillation visited our hospital due to decompensated heart failure. Because atrial fibrillation was considered a remarkable precipitating factor for heart failure, cardioversion was performed. After cardioversion, refractory hypotension and cardiopulmonary arrest occurred. An arterial blood gas analysis showed marked lactic acidosis. Chronic kidney disease, heart failure, sedatives, and hypoventilation might have contributed to refractory hypotension due to severe acidosis in this case.
Assuntos
Fibrilação Atrial/etiologia , Fibrilação Atrial/terapia , Cardioversão Elétrica/efeitos adversos , Parada Cardíaca/etiologia , Insuficiência Cardíaca/complicações , Hipotensão/etiologia , Acidose Láctica , Fibrilação Atrial/fisiopatologia , Gasometria , Feminino , Humanos , Pessoa de Meia-IdadeRESUMO
An 87-year-old man with heart failure was admitted to our hospital. A transthoracic echocardiography showed diffuse mild left ventricular (LV) hypokinesis and LV noncompaction at the apex. A three-dimensional transthoracic echocardiography confirmed a trabecular meshwork. After treatment for heart failure, LV end-systolic dimension decreased and trabeculae seemed to converge and became obscure in end-systole. This is a rare case suggesting mechanism of obscured LV noncompaction after treatment for heart failure.