Detalhe da pesquisa
1.
Complement Destabilizes Cardiomyocyte Function In Vivo after Polymicrobial Sepsis and In Vitro.
J Immunol
; 197(6): 2353-61, 2016 09 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-27521340
2.
Role of extracellular histones in the cardiomyopathy of sepsis.
FASEB J
; 29(5): 2185-93, 2015 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-25681459
3.
Interruption of macrophage-derived IL-27(p28) production by IL-10 during sepsis requires STAT3 but not SOCS3.
J Immunol
; 193(11): 5668-77, 2014 Dec 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-25348624
4.
Changes and regulation of the C5a receptor on neutrophils during septic shock in humans.
J Immunol
; 190(8): 4215-25, 2013 Apr 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-23479227
5.
Inhibition of junctional adhesion molecule-A/LFA interaction attenuates leukocyte trafficking and inflammation in brain ischemia/reperfusion injury.
Neurobiol Dis
; 67: 57-70, 2014 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-24657919
6.
The interaction between C5a and both C5aR and C5L2 receptors is required for production of G-CSF during acute inflammation.
Eur J Immunol
; 43(7): 1907-13, 2013 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-23575697
7.
Regulation of IL-17 family members by adrenal hormones during experimental sepsis in mice.
Am J Pathol
; 182(4): 1124-30, 2013 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-23499051
8.
Extracellular histones are essential effectors of C5aR- and C5L2-mediated tissue damage and inflammation in acute lung injury.
FASEB J
; 27(12): 5010-21, 2013 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-23982144
9.
Complement activation product C5a is a selective suppressor of TLR4-induced, but not TLR3-induced, production of IL-27(p28) from macrophages.
J Immunol
; 188(10): 5086-93, 2012 May 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-22491257
10.
Evidence for anti-inflammatory effects of C5a on the innate IL-17A/IL-23 axis.
FASEB J
; 26(4): 1640-51, 2012 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-22202675
11.
Anti-inflammatory effects of ß2 adrenergic receptor agonists in experimental acute lung injury.
FASEB J
; 26(5): 2137-44, 2012 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-22318967
12.
Generation of C5a in the absence of C3: a new complement activation pathway.
Nat Med
; 12(6): 682-7, 2006 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-16715088
13.
Phagocyte-derived catecholamines enhance acute inflammatory injury.
Nature
; 449(7163): 721-5, 2007 Oct 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-17914358
14.
An essential role for complement C5a in the pathogenesis of septic cardiac dysfunction.
J Exp Med
; 203(1): 53-61, 2006 Jan 23.
Artigo
em Inglês
| MEDLINE | ID: mdl-16380509
15.
An essential role for Stat3 in regulating IgG immune complex-induced pulmonary inflammation.
FASEB J
; 25(12): 4292-300, 2011 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-21859893
16.
Complement dependency of cardiomyocyte release of mediators during sepsis.
FASEB J
; 25(7): 2500-8, 2011 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-21478262
17.
MyD88-dependent production of IL-17F is modulated by the anaphylatoxin C5a via the Akt signaling pathway.
FASEB J
; 25(12): 4222-32, 2011 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-21859896
18.
Cross-talk between TLR4 and FcgammaReceptorIII (CD16) pathways.
PLoS Pathog
; 5(6): e1000464, 2009 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-19503602
19.
The complement system.
Cell Tissue Res
; 343(1): 227-35, 2011 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-20838815
20.
Attenuation of IgG immune complex-induced acute lung injury by silencing C5aR in lung epithelial cells.
FASEB J
; 23(11): 3808-18, 2009 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-19620403