Detalhe da pesquisa
1.
Antiseizure effect of 2-deoxyglucose is not dependent on the presynaptic vacuole ATP pump or the somatic ATP-sensitive K+ channel.
J Neurophysiol
; 129(6): 1423-1433, 2023 06 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-37222440
2.
Increased propensity for infantile spasms and altered neocortical excitation-inhibition balance in a mouse model of down syndrome carrying human chromosome 21.
Neurobiol Dis
; 184: 106198, 2023 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-37315904
3.
Enhanced burst discharges in the CA1 area of the immature versus adult hippocampus: patterns and cellular mechanisms.
J Neurophysiol
; 128(6): 1566-1577, 2022 12 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-36382903
4.
Na+-K+-ATPase functions in the developing hippocampus: regional differences in CA1 and CA3 neuronal excitability and role in epileptiform network bursting.
J Neurophysiol
; 125(1): 1-11, 2021 01 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-33206576
5.
2-Deoxyglucose terminates pilocarpine-induced status epilepticus in neonatal rats.
Epilepsia
; 61(7): 1528-1537, 2020 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-32558935
6.
Glycolytic inhibition by 2-deoxy-d-glucose abolishes both neuronal and network bursts in an in vitro seizure model.
J Neurophysiol
; 118(1): 103-113, 2017 07 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-28404824
7.
Synaptic and extrasynaptic plasticity in glutamatergic circuits involving dentate granule cells following chronic N-methyl-D-aspartate receptor inhibition.
J Neurophysiol
; 109(6): 1535-47, 2013 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-23255721
8.
2-deoxyglucose and ß-hydroxybutyrate fail to attenuate seizures in the betamethasone-NMDA model of infantile spasms.
Epilepsia Open
; 7(1): 181-186, 2022 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-34784103
9.
Repetitive perforant-path stimulation induces epileptiform bursts in minislices of dentate gyrus from rats with kainate-induced epilepsy.
J Neurophysiol
; 105(2): 522-7, 2011 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-21148094
10.
Targeting ischemia-induced KCC2 hypofunction rescues refractory neonatal seizures and mitigates epileptogenesis in a mouse model.
Sci Signal
; 14(708): eabg2648, 2021 Nov 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-34752143
11.
The efficacy of fructose-1,6-bisphosphate in suppressing status epilepticus in developing rats.
Epilepsy Res
; 168: 106500, 2020 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-33161312
12.
Infantile Spasms: An Update on Pre-Clinical Models and EEG Mechanisms.
Children (Basel)
; 7(1)2020 Jan 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-31935804
13.
Both synaptic and intrinsic mechanisms underlie the different properties of population bursts in the hippocampal CA3 area of immature versus adult rats.
J Physiol
; 587(Pt 24): 5907-23, 2009 Dec 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-19884320
14.
2-Deoxyglucose and Beta-Hydroxybutyrate: Metabolic Agents for Seizure Control.
Front Cell Neurosci
; 13: 172, 2019.
Artigo
em Inglês
| MEDLINE | ID: mdl-31114484
15.
Pediatric Epilepsy Mechanisms: Expanding the Paradigm of Excitation/Inhibition Imbalance.
Children (Basel)
; 6(2)2019 Feb 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-30764523
16.
The Glycolytic Metabolite, Fructose-1,6-bisphosphate, Blocks Epileptiform Bursts by Attenuating Voltage-Activated Calcium Currents in Hippocampal Slices.
Front Cell Neurosci
; 12: 168, 2018.
Artigo
em Inglês
| MEDLINE | ID: mdl-29962940
17.
Glycolytic inhibition: A novel approach toward controlling neuronal excitability and seizures.
Epilepsia Open
; 3(Suppl Suppl 2): 191-197, 2018 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-30564778
18.
Pediatric Epileptic Encephalopathies: Pathophysiology and Animal Models.
Semin Pediatr Neurol
; 23(2): 98-107, 2016 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-27544466
19.
Increased Kv1 channel expression may contribute to decreased sIPSC frequency following chronic inhibition of NR2B-containing NMDAR.
Neuropsychopharmacology
; 37(6): 1338-56, 2012 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-22218089
20.
Changes in mIPSCs and sIPSCs after kainate treatment: evidence for loss of inhibitory input to dentate granule cells and possible compensatory responses.
J Neurophysiol
; 94(2): 952-60, 2005 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-15772233