Detalhe da pesquisa
1.
Breaking the oncostatin M feed-forward loop to suppress metastasis and therapy failure.
J Pathol
; 245(1): 6-8, 2018 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-29473175
2.
Oncostatin-M and OSM-Receptor Feed-Forward Activation of MAPK Induces Separable Stem-like and Mesenchymal Programs.
Mol Cancer Res
; 21(9): 975-990, 2023 09 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-37310811
3.
Balancing STAT Activity as a Therapeutic Strategy.
Cancers (Basel)
; 11(11)2019 Nov 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-31684144
4.
A FAM83A Positive Feed-back Loop Drives Survival and Tumorigenicity of Pancreatic Ductal Adenocarcinomas.
Sci Rep
; 9(1): 13396, 2019 09 16.
Artigo
em Inglês
| MEDLINE | ID: mdl-31527715
5.
Cellular plasticity and metastasis in breast cancer: a pre- and post-malignant problem.
J Cancer Metastasis Treat
; 52019.
Artigo
em Inglês
| MEDLINE | ID: mdl-32355893
6.
Targeting Pancreatic Cancer Cell Plasticity: The Latest in Therapeutics.
Cancers (Basel)
; 10(1)2018 Jan 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-29320425
7.
Potent EMT and CSC Phenotypes Are Induced By Oncostatin-M in Pancreatic Cancer.
Mol Cancer Res
; 15(4): 478-488, 2017 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-28053127
8.
Cancer Stem Cell Plasticity Drives Therapeutic Resistance.
Cancers (Basel)
; 8(1)2016 Jan 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-26742077