RESUMO
We describe the first ever-reported occurrence of a post-operative tension pneumosyrinx occurring after a resection of an intradural intramedullary spinal tumour in a 40-year-old patient. Post-operatively, he developed sudden onset paraplegia and imaging revealed a tension pneumosyrinx which was subsequently surgically decompressed. He made a gradual neurological recovery. This is an extremely rare complication with potentially long-lasting deleterious effects on patients' neurological status if not recognized. We aim to bring this pathology to the attention of our neurosurgical colleagues and share our surgical approach and management to assist those who may encounter this pathology in the future.
Assuntos
Procedimentos Neurocirúrgicos , Neoplasias da Medula Espinal , Adulto , Humanos , Doença Iatrogênica , Masculino , Procedimentos Neurocirúrgicos/efeitos adversos , Paraplegia/etiologia , Neoplasias da Medula Espinal/cirurgiaRESUMO
INTRODUCTION: Spinal cord injury is a devastating complication, though rare but possible following the intramuscular injection of the Penicillin. The spinal cord injury can be permanent, leaving the patient with paralysis, bowel and bladder incontinence, and with other associated morbidities. CASE PRESENTATION: We report a 25-year-old gentleman who developed anterior spinal cord syndrome following the benzathine benzylpenicillin injection. In this case report, we discuss the clinical details, possible hypothesis behind spinal cord ischaemia and literature review. DISCUSSION: Spinal cord ischaemia or infarction occurs due to embolism of the Penicillin products. The products following injection are carried as emboli retrogradely through the superior gluteal artery and can cause infarction to the cord's anterior part.
Assuntos
Traumatismos da Medula Espinal , Isquemia do Cordão Espinal , Adulto , Humanos , Infarto/induzido quimicamente , Infarto/etiologia , Injeções Intramusculares/efeitos adversos , Masculino , Penicilina G Benzatina/efeitos adversos , Traumatismos da Medula Espinal/etiologia , Isquemia do Cordão Espinal/induzido quimicamente , Isquemia do Cordão Espinal/complicações , Isquemia do Cordão Espinal/etiologiaRESUMO
Introduction: Chiari malformation is characterized by caudal descent of the cerebellar tonsils through the foramen magnum. Acquired Chiari malformations (ACM) have previously been described after a variety of pathologies including lumbar puncture, cerebrospinal fluid (CSF) drainage, lumboperitoneal shunts, and conditions causing craniocephalic disproportion. Case presentation: We present four cases of ACM following spinal cord injury (SCI), which has not previously been described in the literature. Discussion: ACM is rare and typically associated with abnormalities in CSF pressure or space-occupying lesions. This case series describes the potential association of SCI with ACM. We discuss the imaging findings and clinical management of these patients. Early recognition and intervention may be important to prevent progressive neurology in this vulnerable patient group.
Assuntos
Malformação de Arnold-Chiari/etiologia , Malformação de Arnold-Chiari/patologia , Traumatismos da Medula Espinal/complicações , Traumatismos da Medula Espinal/patologia , Adulto , Pré-Escolar , Humanos , Lactente , Masculino , Adulto JovemRESUMO
INTRODUCTION: Subarachnoid haemorrhage is a catastrophic condition which has significant morbidity and mortality, with mortality rate ranging between 8-67% and is associated with significant morbidity among the survivors. Arachnoiditis of the spinal cord is a very rare complication which can occur months to years after the subarachnoid haemorrhage. CASE PRESENTATION: We report a case of cervicothoracic arachnoiditis following the subarachnoid haemorrhage secondary to posterior inferior cerebellar artery (PICA) aneurysm with posterior fossa AVM (arterio-venous malformation) causing dense paraplegia. DISCUSSION: The haemorrhage that occurs into the fourth ventricle passes to the subarachnoid space via foramen of Magendie and Luschka. This subsequently induces inflammatory changes of the meninges predisposing to arachnoiditis. Early recognition and intervention may prevent this devastating complication or at least reduce the morbidity.