Detalhe da pesquisa
1.
Changes in oxidative stress, inflammation and muscle damage markers following eccentric versus concentric cycling in older adults.
Eur J Appl Physiol
; 119(10): 2301-2312, 2019 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-31451954
2.
Altered ROS production, NF-κB activation and interleukin-6 gene expression induced by electrical stimulation in dystrophic mdx skeletal muscle cells.
Biochim Biophys Acta
; 1852(7): 1410-9, 2015 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-25857619
3.
Mitochondrial Calcium Increase Induced by RyR1 and IP3R Channel Activation After Membrane Depolarization Regulates Skeletal Muscle Metabolism.
Front Physiol
; 9: 791, 2018.
Artigo
em Inglês
| MEDLINE | ID: mdl-29988564
4.
IP3 receptor blockade restores autophagy and mitochondrial function in skeletal muscle fibers of dystrophic mice.
Biochim Biophys Acta Mol Basis Dis
; 1864(11): 3685-3695, 2018 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-30251688
5.
Characterization of a multiprotein complex involved in excitation-transcription coupling of skeletal muscle.
Skelet Muscle
; 6: 15, 2016.
Artigo
em Inglês
| MEDLINE | ID: mdl-27069569
6.
GSK-3ß/NFAT Signaling Is Involved in Testosterone-Induced Cardiac Myocyte Hypertrophy.
PLoS One
; 11(12): e0168255, 2016.
Artigo
em Inglês
| MEDLINE | ID: mdl-27977752
7.
NOX2 Inhibition Impairs Early Muscle Gene Expression Induced by a Single Exercise Bout.
Front Physiol
; 7: 282, 2016.
Artigo
em Inglês
| MEDLINE | ID: mdl-27471471
8.
Electrical stimuli are anti-apoptotic in skeletal muscle via extracellular ATP. Alteration of this signal in Mdx mice is a likely cause of dystrophy.
PLoS One
; 8(11): e75340, 2013.
Artigo
em Inglês
| MEDLINE | ID: mdl-24282497
9.
Nifedipine treatment reduces resting calcium concentration, oxidative and apoptotic gene expression, and improves muscle function in dystrophic mdx mice.
PLoS One
; 8(12): e81222, 2013.
Artigo
em Inglês
| MEDLINE | ID: mdl-24349043
10.
Do adenosine receptors offer new therapeutic options for diabetic nephropathy?
Curr Vasc Pharmacol
; 7(4): 450-9, 2009 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-19485887