Testosterone antagonizes paraquat-induced cardiomyocyte senescence via the mIGF-1/SIRT1 signaling pathway
Yu, Xing; Zheng, Jianyi; Cai, Tengfei; Wang, Zhijian; Zhu, Guiping.
Braz. j. med. biol. res
; 53(10): e9849, 2020. tab, graf
Artigo em Inglês | LILACS, Coleciona SUS (Brasil) | ID: biblio-1132481
Documentos relacionados
Testosterone antagonizes paraquat-induced cardiomyocyte senescence via the mIGF-1/SIRT1 signaling pathway.
"Vermellogens" and the Development of CB[8]-Based Supramolecular Switches Using pH-Responsive and Non-Toxic Viologen Analogues.
[The mechanism of HDAC6 in paraquat-induced autophagy dysfunction of dopaminergic neurons by mediating aggresome-autophagy-lysosomal pathway].
Therapeutic effect of gossypetin against paraquat-induced testicular damage in male rats: a histological and biochemical study.
The Parkinson-Associated Toxin Paraquat Shifts Physiological α-Synuclein Tetramers toward Monomers That Can Be Calpain-Truncated and Form Oligomers.
Testosterone in biosociology: A memoir.
Male reproductive hormone disorders among copper smelter workers.
Phenolic-rich extract of avocado Persea americana (var. Colinred) peel blunts paraquat/maneb-induced apoptosis through blocking phosphorylation of LRRK2 kinase in human nerve-like cells.
Testosterone facilitates nonreproductive, context-appropriate pro- and anti-social behavior in female and male Mongolian gerbils.
Anogenital distance as a toxicological or clinical marker for fetal androgen action and risk for reproductive disorders.