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CD11c-mediated deletion of Flip promotes autoreactivity and inflammatory arthritis.
Huang, Qi-Quan; Perlman, Harris; Birkett, Robert; Doyle, Renee; Fang, Deyu; Haines, G Kenneth; Robinson, William; Datta, Syamal; Huang, Zan; Li, Quan-Zhen; Phee, Hyewon; Pope, Richard M.
Afiliação
  • Huang QQ; Division of Rheumatology, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA.
  • Perlman H; Division of Rheumatology, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA.
  • Birkett R; Division of Rheumatology, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA.
  • Doyle R; Division of Rheumatology, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA.
  • Fang D; Department of Pathology, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA.
  • Haines GK; Department of Pathology, Mount Sinai Hospital School of Medicine, New York city, New York 10029, USA.
  • Robinson W; Division of Immunology and Rheumatology, Department of Medicine, Stanford University School of Medicine, VA Health Care System, Palo Alto, California 94304, USA.
  • Datta S; Division of Rheumatology, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA.
  • Huang Z; Department of Biochemistry, College of Life Sciences, Wuhan University, Wuhan, Hubei 430072, China.
  • Li QZ; Department of Immunology and Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas 75390, USA.
  • Phee H; Department of Microbiology-Immunology, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA.
  • Pope RM; Division of Rheumatology, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA.
Nat Commun ; 6: 7086, 2015 May 12.
Article em En | MEDLINE | ID: mdl-25963626

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Artrite / Antígeno CD11c / Proteína Reguladora de Apoptosis Semelhante a CASP8 e FADD / Inflamação Limite: Animals Idioma: En Revista: Nat Commun Assunto da revista: BIOLOGIA / CIENCIA Ano de publicação: 2015 Tipo de documento: Article País de afiliação: Estados Unidos

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Artrite / Antígeno CD11c / Proteína Reguladora de Apoptosis Semelhante a CASP8 e FADD / Inflamação Limite: Animals Idioma: En Revista: Nat Commun Assunto da revista: BIOLOGIA / CIENCIA Ano de publicação: 2015 Tipo de documento: Article País de afiliação: Estados Unidos