Defining the roles of arrestin2 and arrestin3 in vasoconstrictor receptor desensitization in hypertension.

Willets, Jonathon M; Nash, Craig A; Rainbow, Richard D; Nelson, Carl P; Challiss, R A John

Willets, Jonathon M; Nash, Craig A; Rainbow, Richard D; Nelson, Carl P; Challiss, R A John.

Afiliação

Willets JM; Department of Cell Physiology and Pharmacology, University of Leicester, Leicester, United Kingdom; and jmw23@leicester.ac.uk.
Nash CA; Department of Cell Physiology and Pharmacology, University of Leicester, Leicester, United Kingdom; and.
Rainbow RD; Department of Cardiovascular Sciences, University of Leicester, Glenfield General Hospital, Leicester, United Kingdom.
Nelson CP; Department of Cell Physiology and Pharmacology, University of Leicester, Leicester, United Kingdom; and.
Challiss RA; Department of Cell Physiology and Pharmacology, University of Leicester, Leicester, United Kingdom; and.

Am J Physiol Cell Physiol ; 309(3): C179-89, 2015 Aug 01.

Article em En | MEDLINE | ID: mdl-25972452

Assuntos

Arrestinas/fisiologia; Hipertensão/metabolismo; Vasoconstrição/fisiologia; Animais; Relação Dose-Resposta a Droga; Hipertensão/patologia; Masculino; Artérias Mesentéricas/metabolismo; Artérias Mesentéricas/patologia; Técnicas de Cultura de Órgãos; Ratos; Ratos Endogâmicos SHR; Ratos Endogâmicos WKY; Vasoconstrição/efeitos dos fármacos; Vasoconstritores/farmacologia; beta-Arrestinas

Palavras-chave

G protein-coupled receptor; arrestin; hypertension; phospholipase C; resistance artery; vasoconstrictor

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Vasoconstrição / Arrestinas / Hipertensão Limite: Animals Idioma: En Revista: Am J Physiol Cell Physiol Assunto da revista: FISIOLOGIA Ano de publicação: 2015 Tipo de documento: Article

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