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GRK2 compromises cardiomyocyte mitochondrial function by diminishing fatty acid-mediated oxygen consumption and increasing superoxide levels.
Sato, Priscila Y; Chuprun, J Kurt; Ibetti, Jessica; Cannavo, Alessandro; Drosatos, Konstantinos; Elrod, John W; Koch, Walter J.
Afiliação
  • Sato PY; Center for Translational Medicine & Department of Pharmacology, Temple University School of Medicine, Philadelphia, PA 19140, USA.
  • Chuprun JK; Center for Translational Medicine & Department of Pharmacology, Temple University School of Medicine, Philadelphia, PA 19140, USA.
  • Ibetti J; Center for Translational Medicine & Department of Pharmacology, Temple University School of Medicine, Philadelphia, PA 19140, USA.
  • Cannavo A; Center for Translational Medicine & Department of Pharmacology, Temple University School of Medicine, Philadelphia, PA 19140, USA.
  • Drosatos K; Center for Translational Medicine & Department of Pharmacology, Temple University School of Medicine, Philadelphia, PA 19140, USA.
  • Elrod JW; Center for Translational Medicine & Department of Pharmacology, Temple University School of Medicine, Philadelphia, PA 19140, USA.
  • Koch WJ; Center for Translational Medicine & Department of Pharmacology, Temple University School of Medicine, Philadelphia, PA 19140, USA. Electronic address: Walter.Koch@temple.edu.
J Mol Cell Cardiol ; 89(Pt B): 360-4, 2015 Dec.
Article em En | MEDLINE | ID: mdl-26506135
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Consumo de Oxigênio / Superóxidos / Miócitos Cardíacos / Quinase 2 de Receptor Acoplado a Proteína G / Ácidos Graxos / Mitocôndrias Cardíacas Limite: Animals Idioma: En Revista: J Mol Cell Cardiol Ano de publicação: 2015 Tipo de documento: Article País de afiliação: Estados Unidos
Texto completo
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PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Consumo de Oxigênio / Superóxidos / Miócitos Cardíacos / Quinase 2 de Receptor Acoplado a Proteína G / Ácidos Graxos / Mitocôndrias Cardíacas Limite: Animals Idioma: En Revista: J Mol Cell Cardiol Ano de publicação: 2015 Tipo de documento: Article País de afiliação: Estados Unidos