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Endothelial NOTCH1 is suppressed by circulating lipids and antagonizes inflammation during atherosclerosis.
Briot, Anaïs; Civelek, Mete; Seki, Atsuko; Hoi, Karen; Mack, Julia J; Lee, Stephen D; Kim, Jason; Hong, Cynthia; Yu, Jingjing; Fishbein, Gregory A; Vakili, Ladan; Fogelman, Alan M; Fishbein, Michael C; Lusis, Aldons J; Tontonoz, Peter; Navab, Mohamad; Berliner, Judith A; Iruela-Arispe, M Luisa.
Afiliação
  • Briot A; Department of Molecular, Cell, and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095.
  • Civelek M; Department of Medicine, University of California, Los Angeles, Los Angeles, CA 90095.
  • Seki A; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA 90095.
  • Hoi K; Department of Molecular, Cell, and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095.
  • Mack JJ; Department of Molecular, Cell, and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095.
  • Lee SD; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA 90095 Howard Hughes Medical Institute, Los Angeles, CA 90095.
  • Kim J; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA 90095 Howard Hughes Medical Institute, Los Angeles, CA 90095.
  • Hong C; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA 90095 Howard Hughes Medical Institute, Los Angeles, CA 90095.
  • Yu J; Department of Molecular, Cell, and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095.
  • Fishbein GA; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA 90095.
  • Vakili L; Department of Medicine, University of California, Los Angeles, Los Angeles, CA 90095.
  • Fogelman AM; Department of Medicine, University of California, Los Angeles, Los Angeles, CA 90095.
  • Fishbein MC; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA 90095.
  • Lusis AJ; Department of Medicine, University of California, Los Angeles, Los Angeles, CA 90095.
  • Tontonoz P; Department of Pathology and Laboratory Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA 90095 Howard Hughes Medical Institute, Los Angeles, CA 90095.
  • Navab M; Department of Medicine, University of California, Los Angeles, Los Angeles, CA 90095.
  • Berliner JA; Department of Medicine, University of California, Los Angeles, Los Angeles, CA 90095 Department of Pathology and Laboratory Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA 90095.
  • Iruela-Arispe ML; Department of Molecular, Cell, and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095 Molecular Biology Institute, University of California, Los Angeles, Los Angeles, CA 90095 arispe@mcdb.ucla.edu.
J Exp Med ; 212(12): 2147-63, 2015 Nov 16.
Article em En | MEDLINE | ID: mdl-26552708
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Células Endoteliais / Aterosclerose / Receptor Notch1 / Inflamação / Lipídeos Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Adult / Animals / Female / Humans / Male Idioma: En Revista: J Exp Med Ano de publicação: 2015 Tipo de documento: Article
Texto completo
Imprimir
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PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Células Endoteliais / Aterosclerose / Receptor Notch1 / Inflamação / Lipídeos Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Adult / Animals / Female / Humans / Male Idioma: En Revista: J Exp Med Ano de publicação: 2015 Tipo de documento: Article