Air pollutant-mediated disruption of sinonasal epithelial cell barrier function is reversed by activation of the Nrf2 pathway.

London, Nyall R; Tharakan, Anuj; Rule, Ana M; Lane, Andrew P; Biswal, Shyam; Ramanathan, Murugappan

London, Nyall R; Tharakan, Anuj; Rule, Ana M; Lane, Andrew P; Biswal, Shyam; Ramanathan, Murugappan.

Afiliação

London NR; Johns Hopkins Department of Otolaryngology-Head and Neck Surgery, Baltimore, Md.
Tharakan A; Johns Hopkins Department of Otolaryngology-Head and Neck Surgery, Baltimore, Md.
Rule AM; Department of Environmental Health Sciences, Johns Hopkins Bloomberg School of Public Health, Baltimore, Md.
Lane AP; Johns Hopkins Department of Otolaryngology-Head and Neck Surgery, Baltimore, Md.
Biswal S; Department of Environmental Health Sciences, Johns Hopkins Bloomberg School of Public Health, Baltimore, Md.
Ramanathan M; Johns Hopkins Department of Otolaryngology-Head and Neck Surgery, Baltimore, Md. Electronic address: mramana3@jhmi.edu.

J Allergy Clin Immunol ; 138(6): 1736-1738.e4, 2016 12.

Article em En | MEDLINE | ID: mdl-27576127

Assuntos

Poluentes Atmosféricos/efeitos adversos; Poluição do Ar/efeitos adversos; Células Epiteliais/efeitos dos fármacos; Células Epiteliais/metabolismo; Fator 2 Relacionado a NF-E2/metabolismo; Animais; Humanos; Material Particulado/efeitos adversos; Proteína da Zônula de Oclusão-1/metabolismo

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Poluentes Atmosféricos / Poluição do Ar / Células Epiteliais / Fator 2 Relacionado a NF-E2 Limite: Animals / Humans Idioma: En Revista: J Allergy Clin Immunol Ano de publicação: 2016 Tipo de documento: Article País de afiliação: Moldávia

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