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CXCR3 regulates CD4+ T cell cardiotropism in pressure overload-induced cardiac dysfunction.
Ngwenyama, Njabulo; Salvador, Ane M; Velázquez, Francisco; Nevers, Tania; Levy, Alexander; Aronovitz, Mark; Luster, Andrew D; Huggins, Gordon S; Alcaide, Pilar.
Afiliação
  • Ngwenyama N; Department of Immunology and.
  • Salvador AM; Department of Immunology and.
  • Velázquez F; Department of Immunology and.
  • Nevers T; Department of Immunology and.
  • Levy A; Molecular Cardiology Research Institute Tufts University, Boston, Massachusetts, USA.
  • Aronovitz M; Molecular Cardiology Research Institute Tufts University, Boston, Massachusetts, USA.
  • Luster AD; Division of Rheumatology, Allergy and Immunology, Center for Immunology and Inflammatory Diseases, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, USA.
  • Huggins GS; Molecular Cardiology Research Institute Tufts University, Boston, Massachusetts, USA.
  • Alcaide P; Department of Immunology and.
JCI Insight ; 4(7)2019 04 04.
Article em En | MEDLINE | ID: mdl-30779709

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Células Th1 / Receptores CXCR3 / Insuficiência Cardíaca / Miocárdio Tipo de estudo: Diagnostic_studies / Prognostic_studies Limite: Animals / Humans / Male Idioma: En Revista: JCI Insight Ano de publicação: 2019 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Células Th1 / Receptores CXCR3 / Insuficiência Cardíaca / Miocárdio Tipo de estudo: Diagnostic_studies / Prognostic_studies Limite: Animals / Humans / Male Idioma: En Revista: JCI Insight Ano de publicação: 2019 Tipo de documento: Article