IL-1&#945; and IL-1ß promote NOD2-induced immune responses by enhancing MAPK signaling.

Li, Sushan; Deng, Ping; Wang, Manzhi; Liu, Xueting; Jiang, Manli; Jiang, Binyuan; Yang, Li; Hu, Jinyue

IL-1α and IL-1ß promote NOD2-induced immune responses by enhancing MAPK signaling.

Li, Sushan; Deng, Ping; Wang, Manzhi; Liu, Xueting; Jiang, Manli; Jiang, Binyuan; Yang, Li; Hu, Jinyue.

Afiliação

Li S; Department of Cardiology, Changsha Central Hospital, Changsha, China.
Deng P; Graduate School, University of South China, Hengyang, China.
Wang M; Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, China.
Liu X; Department of Cardiology, Changsha Central Hospital, Changsha, China. 1229015862@qq.com.
Jiang M; Department of Pediatrics, Changsha Central Hospital, Changsha, China.
Jiang B; Medical Research Center, Changsha Central Hospital, Changsha, China.
Yang L; Medical Research Center, Changsha Central Hospital, Changsha, China.
Hu J; Medical Research Center, Changsha Central Hospital, Changsha, China.

Lab Invest ; 99(9): 1321-1334, 2019 09.

Article em En | MEDLINE | ID: mdl-31019287

Assuntos

Imunidade Inata/fisiologia; Interleucina-1alfa/metabolismo; Interleucina-1beta/metabolismo; Proteínas Quinases Ativadas por Mitógeno/metabolismo; Proteína Adaptadora de Sinalização NOD2/metabolismo; Animais; Citocinas/metabolismo; Humanos; Masculino; Camundongos; Camundongos Endogâmicos C57BL; Células RAW 264.7; Células THP-1

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteínas Quinases Ativadas por Mitógeno / Interleucina-1alfa / Interleucina-1beta / Proteína Adaptadora de Sinalização NOD2 / Imunidade Inata Limite: Animals / Humans / Male Idioma: En Revista: Lab Invest Ano de publicação: 2019 Tipo de documento: Article País de afiliação: China

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