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Inducing and exploiting vulnerabilities for the treatment of liver cancer.
Wang, Cun; Vegna, Serena; Jin, Haojie; Benedict, Bente; Lieftink, Cor; Ramirez, Christel; de Oliveira, Rodrigo Leite; Morris, Ben; Gadiot, Jules; Wang, Wei; du Chatinier, Aimée; Wang, Liqin; Gao, Dongmei; Evers, Bastiaan; Jin, Guangzhi; Xue, Zheng; Schepers, Arnout; Jochems, Fleur; Sanchez, Antonio Mulero; Mainardi, Sara; Te Riele, Hein; Beijersbergen, Roderick L; Qin, Wenxin; Akkari, Leila; Bernards, René.
Afiliação
  • Wang C; State Key Laboratory of Oncogenes and Related Genes, Shanghai Cancer Institute, Renji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
  • Vegna S; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Jin H; Oncode Institute, Division of Tumour Biology and Immunology, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Benedict B; State Key Laboratory of Oncogenes and Related Genes, Shanghai Cancer Institute, Renji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
  • Lieftink C; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Ramirez C; Oncode Institute, Division of Tumour Biology and Immunology, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • de Oliveira RL; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Morris B; Oncode Institute, Division of Tumour Biology and Immunology, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Gadiot J; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Wang W; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • du Chatinier A; Oncode Institute, Division of Tumour Biology and Immunology, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Wang L; Division of Cell Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Gao D; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Evers B; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Jin G; Liver Cancer Institute, Zhongshan Hospital, Fudan University, Key Laboratory of Carcinogenesis and Cancer Invasion, Ministry of Education, Shanghai, China.
  • Xue Z; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Schepers A; Department of Pathology, Eastern Hepatobiliary Surgery Hospital, Second Military Medical University, Shanghai, China.
  • Jochems F; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Sanchez AM; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Mainardi S; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Te Riele H; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Beijersbergen RL; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Qin W; Oncode Institute, Division of Tumour Biology and Immunology, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Akkari L; Oncode Institute, Division of Molecular Carcinogenesis, The Netherlands Cancer Institute, Amsterdam, The Netherlands.
  • Bernards R; State Key Laboratory of Oncogenes and Related Genes, Shanghai Cancer Institute, Renji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China. wxqin@sjtu.edu.cn.
Nature ; 574(7777): 268-272, 2019 10.
Article em En | MEDLINE | ID: mdl-31578521
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Senescência Celular / Apoptose / Sertralina / Terapia de Alvo Molecular / Neoplasias Hepáticas Limite: Animals Idioma: En Revista: Nature Ano de publicação: 2019 Tipo de documento: Article País de afiliação: China
Texto completo
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XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Senescência Celular / Apoptose / Sertralina / Terapia de Alvo Molecular / Neoplasias Hepáticas Limite: Animals Idioma: En Revista: Nature Ano de publicação: 2019 Tipo de documento: Article País de afiliação: China