Role of IL-25, IL-33, and TSLP in triggering united airway diseases toward type 2 inflammation.

Hong, Haiyu; Liao, Shumin; Chen, Fenghong; Yang, Qintai; Wang, De-Yun

Hong, Haiyu; Liao, Shumin; Chen, Fenghong; Yang, Qintai; Wang, De-Yun.

Afiliação

Hong H; Department of Otolaryngology, Allergy Center, The Fifth Affiliated Hospital of Sun Yat-sen University, Zhuhai, China.
Liao S; Department of Otolaryngology, National University of Singapore, National University Health System, Singapore, Singapore.
Chen F; Department of Otolaryngology, Allergy Center, The Fifth Affiliated Hospital of Sun Yat-sen University, Zhuhai, China.
Yang Q; Otorhinolaryngology Hospital, The First Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Wang DY; Department of Otolaryngology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.

Allergy ; 75(11): 2794-2804, 2020 11.

Article em En | MEDLINE | ID: mdl-32737888

Assuntos

Asma; Rinite Alérgica; Sinusite; Asma/epidemiologia; Asma/etiologia; Citocinas; Humanos; Inflamação; Interleucina-33; Sinusite/etiologia

Palavras-chave

IL-25; IL-33; TSLP; type 2 inflammation; united airway disease

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Asma / Sinusite / Rinite Alérgica Tipo de estudo: Etiology_studies Limite: Humans Idioma: En Revista: Allergy Ano de publicação: 2020 Tipo de documento: Article País de afiliação: China

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