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Liposomal UHRF1 siRNA shows lung fibrosis treatment potential through regulation of fibroblast activation.
Cheng, Demin; Wang, Yue; Li, Ziwei; Xiong, Haojie; Sun, Wenqing; Xi, Sichuan; Zhou, Siyun; Liu, Yi; Ni, Chunhui.
Afiliação
  • Cheng D; Department of Occupational Medical and Environmental Health, Key Laboratory of Modern Toxicology, Ministry of Education, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, China.
  • Wang Y; Department of Occupational Medical and Environmental Health, Key Laboratory of Modern Toxicology, Ministry of Education, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, China.
  • Li Z; Department of Occupational Medical and Environmental Health, Key Laboratory of Modern Toxicology, Ministry of Education, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, China.
  • Xiong H; Department of Occupational Medical and Environmental Health, Key Laboratory of Modern Toxicology, Ministry of Education, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, China.
  • Sun W; Department of Occupational Medical and Environmental Health, Key Laboratory of Modern Toxicology, Ministry of Education, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, China.
  • Xi S; Thoracic Epigenetics Section, Thoracic Surgery Branch, National Cancer Institute, NIH, Bethesda, Maryland, USA.
  • Zhou S; Department of Occupational Medical and Environmental Health, Key Laboratory of Modern Toxicology, Ministry of Education, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, China.
  • Liu Y; Gusu School, Nanjing Medical University, Nanjing, China.
  • Ni C; Department of Occupational Medical and Environmental Health, Key Laboratory of Modern Toxicology, Ministry of Education, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, China.
JCI Insight ; 7(22)2022 11 22.
Article em En | MEDLINE | ID: mdl-36166308
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Fibrose Pulmonar / Proteínas Estimuladoras de Ligação a CCAAT Limite: Animals Idioma: En Revista: JCI Insight Ano de publicação: 2022 Tipo de documento: Article País de afiliação: China
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Fibrose Pulmonar / Proteínas Estimuladoras de Ligação a CCAAT Limite: Animals Idioma: En Revista: JCI Insight Ano de publicação: 2022 Tipo de documento: Article País de afiliação: China