MK2 Promotes the Development and Progression of Pancreatic Neuroendocrine Tumors Mediated by Macrophages and Metabolomic Factors.

Jacenik, Damian; Lebish, Eric J; Beswick, Ellen J

Jacenik, Damian; Lebish, Eric J; Beswick, Ellen J.

Afiliação

Jacenik D; Department of Cytobiochemistry, Faculty of Biology and Environmental Protection, University of Lodz, 90-236 Lodz, Poland.
Lebish EJ; Division of Gastroenterology, Hepatology and Nutrition, Department of Internal Medicine, University of Utah, Salt Lake City, UT 84132, USA.
Beswick EJ; Division of Gastroenterology, Hepatology and Nutrition, Department of Internal Medicine, University of Utah, Salt Lake City, UT 84132, USA.

Int J Mol Sci ; 23(21)2022 Nov 05.

Article em En | MEDLINE | ID: mdl-36362348

Assuntos

Tumores Neuroectodérmicos Primitivos; Tumores Neuroendócrinos; Neoplasias Pancreáticas; Animais; Camundongos; Tumores Neuroendócrinos/metabolismo; Modelos Animais de Doenças; Macrófagos/metabolismo; Citocinas/metabolismo; Quimiocinas/metabolismo; Neoplasias Pancreáticas/metabolismo; Tumores Neuroectodérmicos Primitivos/metabolismo

Palavras-chave

MAPKAPK2; MK2; mitogen-activated protein kinase-activated protein kinase 2; neuroendocrine tumor; pancreas; pancreatic neuroendocrine tumor

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Neoplasias Pancreáticas / Tumores Neuroendócrinos / Tumores Neuroectodérmicos Primitivos Tipo de estudo: Prognostic_studies Limite: Animals Idioma: En Revista: Int J Mol Sci Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Polônia

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