Detalhe da pesquisa
1.
Growth hormone-releasing hormone agonists ameliorate chronic kidney disease-induced heart failure with preserved ejection fraction.
Proc Natl Acad Sci U S A
; 118(4)2021 01 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-33468654
2.
Efficacy of a growth hormone-releasing hormone agonist in a murine model of cardiometabolic heart failure with preserved ejection fraction.
Am J Physiol Heart Circ Physiol
; 324(6): H739-H750, 2023 06 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-36897749
3.
NOX Inhibition Improves ß-Adrenergic Stimulated Contractility and Intracellular Calcium Handling in the Aged Rat Heart.
Int J Mol Sci
; 19(8)2018 Aug 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-30111689
4.
Interaction between neuronal nitric oxide synthase signaling and temperature influences sarcoplasmic reticulum calcium leak: role of nitroso-redox balance.
Circ Res
; 116(1): 46-55, 2015 Jan 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-25326127
5.
S-nitrosoglutathione reductase (GSNOR) enhances vasculogenesis by mesenchymal stem cells.
Proc Natl Acad Sci U S A
; 110(8): 2834-9, 2013 Feb 19.
Artigo
em Inglês
| MEDLINE | ID: mdl-23288904
6.
Long term ablation of protein kinase A (PKA)-mediated cardiac troponin I phosphorylation leads to excitation-contraction uncoupling and diastolic dysfunction in a knock-in mouse model of hypertrophic cardiomyopathy.
J Biol Chem
; 289(33): 23097-23111, 2014 Aug 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-24973218
7.
Dynamic denitrosylation via S-nitrosoglutathione reductase regulates cardiovascular function.
Proc Natl Acad Sci U S A
; 109(11): 4314-9, 2012 Mar 13.
Artigo
em Inglês
| MEDLINE | ID: mdl-22366318
8.
Hydralazine and organic nitrates restore impaired excitation-contraction coupling by reducing calcium leak associated with nitroso-redox imbalance.
J Biol Chem
; 288(9): 6522-33, 2013 Mar 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-23319593
9.
NADPH oxidase-2 inhibition restores contractility and intracellular calcium handling and reduces arrhythmogenicity in dystrophic cardiomyopathy.
Am J Physiol Heart Circ Physiol
; 307(5): H710-21, 2014 Sep 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-25015966
10.
C-kit(+) cells isolated from developing kidneys are a novel population of stem cells with regenerative potential.
Stem Cells
; 31(8): 1644-56, 2013 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-23733311
11.
Synthetic growth hormone-releasing hormone agonist ameliorates the myocardial pathophysiology characteristic of heart failure with preserved ejection fraction.
Cardiovasc Res
; 118(18): 3586-3601, 2023 02 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-35704032
12.
S-Nitrosoglutathione Reductase Deficiency Causes Aberrant Placental S-Nitrosylation and Preeclampsia.
J Am Heart Assoc
; 11(5): e024008, 2022 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-35191317
13.
Impaired S-nitrosylation of the ryanodine receptor caused by xanthine oxidase activity contributes to calcium leak in heart failure.
J Biol Chem
; 285(37): 28938-45, 2010 Sep 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-20643651
14.
S-nitrosoglutathione reductase (GSNOR) deficiency accelerates cardiomyocyte differentiation of induced pluripotent stem cells.
J Cardiovasc Aging
; 12021.
Artigo
em Inglês
| MEDLINE | ID: mdl-34790975
15.
Early signals after stretch leading to cardiac hypertrophy. Key role of NHE-1.
Front Biosci
; 13: 7096-114, 2008 May 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-18508719
16.
S-glutathionylation: a redox-sensitive switch participating in nitroso-redox balance.
Circ Res
; 108(5): 531-3, 2011 Mar 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-21372289
17.
Wnt signalling: a mediator of the heart-bone marrow axis after myocardial injury?
Eur Heart J
; 33(15): 1861-3, 2012 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-22199122
18.
Influence of Na+-independent Cl--HCO3- exchange on the slow force response to myocardial stretch.
Circ Res
; 93(11): 1082-8, 2003 Nov 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-14576196
19.
Endothelin 1 versus endothelin 3 in the development of the slow force response to myocardial stretch.
Can J Cardiol
; 21(5): 435-8, 2005 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-15861262
20.
A low dose of angiotensin II increases inotropism through activation of reverse Na(+)/Ca(2+) exchange by endothelin release.
Cardiovasc Res
; 60(3): 589-97, 2003 Dec 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-14659804