Detalhe da pesquisa
1.
CUG-BP, Elav-like family member 1 (CELF1) is required for normal myofibrillogenesis, morphogenesis, and contractile function in the embryonic heart.
Dev Dyn
; 245(8): 854-73, 2016 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-27144987
2.
Muscleblind-like 1 is required for normal heart valve development in vivo.
BMC Dev Biol
; 15: 36, 2015 Oct 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-26472242
3.
CUG-BP, Elav-like family (CELF)-mediated alternative splicing regulation in the brain during health and disease.
Mol Cell Neurosci
; 56: 456-64, 2013 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-23247071
4.
Loss of muscleblind-like 1 promotes invasive mesenchyme formation in endocardial cushions by stimulating autocrine TGFß3.
BMC Dev Biol
; 12: 22, 2012 Aug 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-22866814
5.
Alternative splicing of the neurofibromatosis type 1 pre-mRNA is regulated by the muscleblind-like proteins and the CUG-BP and ELAV-like factors.
BMC Mol Biol
; 13: 35, 2012 Dec 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-23227900
6.
The neurofibromatosis type I pre-mRNA is a novel target of CELF protein-mediated splicing regulation.
Nucleic Acids Res
; 38(1): 253-64, 2010 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-19854948
7.
Differential susceptibility of muscles to myotonia and force impairment in a mouse model of myotonic dystrophy.
Muscle Nerve
; 43(6): 818-27, 2011 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-21404300
8.
Conserved developmental alternative splicing of muscleblind-like (MBNL) transcripts regulates MBNL localization and activity.
RNA Biol
; 7(1): 43-55, 2010.
Artigo
em Inglês
| MEDLINE | ID: mdl-20009516
9.
Muscleblind-like 1 is a negative regulator of TGF-beta-dependent epithelial-mesenchymal transition of atrioventricular canal endocardial cells.
Dev Dyn
; 238(12): 3266-72, 2009 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-19890912
10.
CELF-mediated alternative splicing is required for cardiac function during early, but not later, postnatal life.
J Mol Cell Cardiol
; 46(3): 395-404, 2009 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-19073192
11.
Known turnover and translation regulatory RNA-binding proteins interact with the 3' UTR of SECIS-binding protein 2.
RNA Biol
; 6(1): 73-83, 2009.
Artigo
em Inglês
| MEDLINE | ID: mdl-19106619
12.
Cardiac tissue-specific repression of CELF activity disrupts alternative splicing and causes cardiomyopathy.
Mol Cell Biol
; 25(14): 6267-78, 2005 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-15988035
13.
Identification of Targets of CUG-BP, Elav-Like Family Member 1 (CELF1) Regulation in Embryonic Heart Muscle.
PLoS One
; 11(2): e0149061, 2016.
Artigo
em Inglês
| MEDLINE | ID: mdl-26866591
14.
Identification of transcripts regulated by CUG-BP, Elav-like family member 1 (CELF1) in primary embryonic cardiomyocytes by RNA-seq.
Genom Data
; 6: 74-76, 2015 Dec 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-26366374
15.
Gene Expression Analyses during Spontaneous Reversal of Cardiomyopathy in Mice with Repressed Nuclear CUG-BP, Elav-Like Family (CELF) Activity in Heart Muscle.
PLoS One
; 10(4): e0124462, 2015.
Artigo
em Inglês
| MEDLINE | ID: mdl-25894229
16.
RNA binding proteins in the regulation of heart development.
Int J Biochem Cell Biol
; 45(11): 2467-78, 2013 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-23973289
17.
Gene expression analyses implicate an alternative splicing program in regulating contractile gene expression and serum response factor activity in mice.
PLoS One
; 8(2): e56590, 2013.
Artigo
em Inglês
| MEDLINE | ID: mdl-23437181
18.
The importance of CELF control: molecular and biological roles of the CUG-BP, Elav-like family of RNA-binding proteins.
Wiley Interdiscip Rev RNA
; 3(1): 104-21, 2012.
Artigo
em Inglês
| MEDLINE | ID: mdl-22180311
19.
Repression of nuclear CELF activity can rescue CELF-regulated alternative splicing defects in skeletal muscle models of myotonic dystrophy.
PLoS Curr
; 4: RRN1305, 2012 Feb 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-22453899
20.
Expression of a dominant negative CELF protein in vivo leads to altered muscle organization, fiber size, and subtype.
PLoS One
; 6(4): e19274, 2011 Apr 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-21541285