Detalhe da pesquisa
1.
CD36 as a gatekeeper of myocardial lipid metabolism and therapeutic target for metabolic disease.
Physiol Rev
; 104(2): 727-764, 2024 Apr 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-37882731
2.
Glycolysis-Mediated Activation of v-ATPase by Nicotinamide Mononucleotide Ameliorates Lipid-Induced Cardiomyopathy by Repressing the CD36-TLR4 Axis.
Circ Res
; 134(5): 505-525, 2024 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-38422177
3.
CD36 (SR-B2) as master regulator of cellular fatty acid homeostasis.
Curr Opin Lipidol
; 33(2): 103-111, 2022 04 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-35125400
4.
Ketone Body Exposure of Cardiomyocytes Impairs Insulin Sensitivity and Contractile Function through Vacuolar-Type H+-ATPase Disassembly-Rescue by Specific Amino Acid Supplementation.
Int J Mol Sci
; 23(21)2022 Oct 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-36361698
5.
Metabolic Interventions to Prevent Hypertrophy-Induced Alterations in Contractile Properties In Vitro.
Int J Mol Sci
; 22(7)2021 Mar 31.
Artigo
em Inglês
| MEDLINE | ID: mdl-33807195
6.
Putative Role of Protein Palmitoylation in Cardiac Lipid-Induced Insulin Resistance.
Int J Mol Sci
; 21(24)2020 Dec 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-33322406
7.
Augmenting Vacuolar H+-ATPase Function Prevents Cardiomyocytes from Lipid-Overload Induced Dysfunction.
Int J Mol Sci
; 21(4)2020 Feb 23.
Artigo
em Inglês
| MEDLINE | ID: mdl-32102213
8.
Dynamic role of the transmembrane glycoprotein CD36 (SR-B2) in cellular fatty acid uptake and utilization.
J Lipid Res
; 59(7): 1084-1093, 2018 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-29627764
9.
2-Arachidonoylglycerol ameliorates inflammatory stress-induced insulin resistance in cardiomyocytes.
J Biol Chem
; 292(17): 7105-7114, 2017 04 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-28320859
10.
Human embryonic stem cell-derived cardiomyocytes as an in vitro model to study cardiac insulin resistance.
Biochim Biophys Acta Mol Basis Dis
; 1864(5 Pt B): 1960-1967, 2018 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-29277329
11.
A new leptin-mediated mechanism for stimulating fatty acid oxidation: a pivotal role for sarcolemmal FAT/CD36.
Biochem J
; 474(1): 149-162, 2017 01 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-27827305
12.
Regulation of the subcellular trafficking of CD36, a major determinant of cardiac fatty acid utilization.
Biochim Biophys Acta
; 1861(10): 1461-71, 2016 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-27090938
13.
Post-translational modifications of CD36 (SR-B2): Implications for regulation of myocellular fatty acid uptake.
Biochim Biophys Acta
; 1862(12): 2253-2258, 2016 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-27615427
14.
Membrane fatty acid transporters as regulators of lipid metabolism: implications for metabolic disease.
Physiol Rev
; 90(1): 367-417, 2010 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-20086080
15.
The interaction between AMPKß2 and the PP1-targeting subunit R6 is dynamically regulated by intracellular glycogen content.
Biochem J
; 473(7): 937-47, 2016 Apr 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-26831516
16.
Marine omega-3 fatty acids prevent myocardial insulin resistance and metabolic remodeling as induced experimentally by high insulin exposure.
Am J Physiol Cell Physiol
; 308(4): C297-307, 2015 Feb 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-25472960
17.
Time for a détente in the war on the mechanism of cellular fatty acid uptake.
J Lipid Res
; 61(9): 1300-1303, 2020 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-32873748
18.
Extremely rapid increase in fatty acid transport and intramyocellular lipid accumulation but markedly delayed insulin resistance after high fat feeding in rats.
Diabetologia
; 58(10): 2381-91, 2015 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-26197708
19.
Cardiac diastolic dysfunction in high-fat diet fed mice is associated with lipotoxicity without impairment of cardiac energetics in vivo.
Biochim Biophys Acta
; 1842(10): 1525-37, 2014 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-25086219
20.
Protein kinase-D1 overexpression prevents lipid-induced cardiac insulin resistance.
J Mol Cell Cardiol
; 76: 208-17, 2014 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-25173922