Detalhe da pesquisa
1.
Glycolytic-to-oxidative fiber-type switch and mTOR signaling activation are early-onset features of SBMA muscle modified by high-fat diet.
Acta Neuropathol
; 132(1): 127-44, 2016 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-26971100
2.
p66Shc deletion or deficiency protects from obesity but not metabolic dysfunction in mice and humans.
Diabetologia
; 58(10): 2352-60, 2015 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-26122877
3.
KBTBD13 is an actin-binding protein that modulates muscle kinetics.
J Clin Invest
; 130(2): 754-767, 2020 02 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-31671076
4.
KBTBD13 is an actin-binding protein that modulates muscle kinetics.
J Clin Invest
; 134(3)2024 02 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-38299595
5.
Drug Repurposing for Duchenne Muscular Dystrophy: The Monoamine Oxidase B Inhibitor Safinamide Ameliorates the Pathological Phenotype in mdx Mice and in Myogenic Cultures From DMD Patients.
Front Physiol
; 9: 1087, 2018.
Artigo
em Inglês
| MEDLINE | ID: mdl-30154729
6.
Comparative Analysis of Muscle Hypertrophy Models Reveals Divergent Gene Transcription Profiles and Points to Translational Regulation of Muscle Growth through Increased mTOR Signaling.
Front Physiol
; 8: 968, 2017.
Artigo
em Inglês
| MEDLINE | ID: mdl-29255421
7.
S6K1 Is Required for Increasing Skeletal Muscle Force during Hypertrophy.
Cell Rep
; 17(2): 501-513, 2016 10 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-27705797