Detalhe da pesquisa
1.
Drugs that inhibit TMEM16 proteins block SARS-CoV-2 spike-induced syncytia.
Nature
; 594(7861): 88-93, 2021 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-33827113
2.
Human blood vessel organoids reveal a critical role for CTGF in maintaining microvascular integrity.
Nat Commun
; 14(1): 5552, 2023 09 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-37689702
3.
SARS-CoV-2 Spike protein activates TMEM16F-mediated platelet procoagulant activity.
Front Cardiovasc Med
; 9: 1013262, 2022.
Artigo
em Inglês
| MEDLINE | ID: mdl-36684586
4.
Combined in silico and in vitro approaches identified the antipsychotic drug lurasidone and the antiviral drug elbasvir as SARS-CoV2 and HCoV-OC43 inhibitors.
Antiviral Res
; 189: 105055, 2021 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-33713730
5.
SARS-CoV-2, myocardial injury and inflammation: insights from a large clinical and autopsy study.
Clin Res Cardiol
; 110(11): 1822-1831, 2021 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-34282465
6.
CRISPR-Csy4-Mediated Editing of Rotavirus Double-Stranded RNA Genome.
Cell Rep
; 32(13): 108205, 2020 09 29.
Artigo
em Inglês
| MEDLINE | ID: mdl-32997981
7.
Persistence of viral RNA, pneumocyte syncytia and thrombosis are hallmarks of advanced COVID-19 pathology.
EBioMedicine
; 61: 103104, 2020 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-33158808
8.
Correction to: SARSCoV2, myocardial injury and inflammation: insights from a large clinical and autopsy study.
Clin Res Cardiol
; 110(10): 1694, 2021 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-34398286