Papel da Stress Inducible Phosphoprotein One (STI1) e da proteína prion celular na angiogênese tumoral

Cartaxo, Rodrigo Teodoro.
São Paulo; s.n; 2018. 237 p. ilust.
Tese em Português | LILACS, Inca | ID: biblio-1255269

Documentos relacionados

  1. Analysis of co-isogenic prion protein deficient mice reveals behavioral deficits, learning impairment, and enhanced hippocampal excitability.
  2. The protean prion protein.
  3. Effects of prion protein devoid of the N-terminal residues 25-50 on prion pathogenesis in mice.
  4. <i>In Vitro</i> Approach To Identify Key Amino Acids in Low Susceptibility of Rabbit Prion Protein to Misfolding.
  5. Central residues in prion protein PrP<sup>C</sup> are crucial for its conversion into the pathogenic isoform.
  6. Efficient transmission of human prion diseases to a glycan-free prion protein-expressing host.
  7. Heterozygosity for cervid S138N polymorphism results in subclinical CWD in gene-targeted mice and progressive inhibition of prion conversion.
  8. New and distinct chronic wasting disease strains associated with cervid polymorphism at codon 116 of the Prnp gene.
  9. Condition-dependent structural collapse in the intrinsically disordered N-terminal domain of prion protein.
  10. Cellular Prion Protein Mediates &#945;-Synuclein Uptake, Localization, and Toxicity In Vitro and In Vivo.