Pathological impact of hyperpolarization-activated chloride current peculiar to rat pulmonary vein cardiomyocytes.
J Mol Cell Cardiol
; 66: 53-62, 2014 Jan.
Article
em En
| MEDLINE
| ID: mdl-24239603
Palavras-chave
4,4'-diisothiocyanostilbene-2,2'-disulfonic acid; 9-AC; 9-anthracene carboxylic acid; AF; Atrial fibrillation; Ca(2+) activated Cl(−) channel; Cl(−) channel; Cm; DIDS; GAPDH; I(ClCa); I(K1); I(KH); I(f); I(ti); LA; NE; PV; Pulmonary vein; atrial fibrillation; cell membrane capacitance; glyceraldehyde-3-phosphate dehydrogenase; hyperpolarization-activated K(+) current; hyperpolarization-activated cation current; inward-rectifier K(+) current; left atrium; norepinephrine; pulmonary vein; qPCR; quantitative polymerase chain reaction; transient inward current
Texto completo:
1
Base de dados:
MEDLINE
Assunto principal:
Veias Pulmonares
/
Cloretos
/
Canais de Cloreto
/
Miócitos Cardíacos
Limite:
Animals
Idioma:
En
Revista:
J Mol Cell Cardiol
Ano de publicação:
2014
Tipo de documento:
Article
País de afiliação:
Japão