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Gain of function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies.
Yang, Hui; Kurtenbach, Stefan; Guo, Ying; Lohse, Ines; Durante, Michael A; Li, Jianping; Li, Zhaomin; Al-Ali, Hassan; Li, Lingxiao; Chen, Zizhen; Field, Matthew G; Zhang, Peng; Chen, Shi; Yamamoto, Shohei; Li, Zhuo; Zhou, Yuan; Nimer, Stephen D; Harbour, J William; Wahlestedt, Claes; Xu, Mingjiang; Yang, Feng-Chun.
Afiliação
  • Yang H; Sylvester Comprehensive Cancer Center.
  • Kurtenbach S; Department of Biochemistry and Molecular Biology.
  • Guo Y; Sylvester Comprehensive Cancer Center.
  • Lohse I; Bascom Palmer Eye Institute.
  • Durante MA; Sylvester Comprehensive Cancer Center.
  • Li J; Department of Biochemistry and Molecular Biology.
  • Li Z; Sylvester Comprehensive Cancer Center.
  • Al-Ali H; Center for Therapeutic Innovation and Department of Psychiatry and Behavioral Sciences.
  • Li L; Sylvester Comprehensive Cancer Center.
  • Chen Z; Bascom Palmer Eye Institute.
  • Field MG; Sylvester Comprehensive Cancer Center.
  • Zhang P; Department of Biochemistry and Molecular Biology.
  • Chen S; Sylvester Comprehensive Cancer Center.
  • Yamamoto S; Department of Biochemistry and Molecular Biology.
  • Li Z; Sylvester Comprehensive Cancer Center.
  • Zhou Y; Department of Neurological Surgery, The Miami Project to Cure Paralysis, Peggy and Harold Katz Family Drug Discovery Center, and.
  • Nimer SD; Sylvester Comprehensive Cancer Center.
  • Harbour JW; Department of Internal Medicine, University of Miami Miller School of Medicine, Miami, FL; and.
  • Wahlestedt C; State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital and Center for Stem Cell Medicine, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
  • Xu M; Sylvester Comprehensive Cancer Center.
  • Yang FC; Bascom Palmer Eye Institute.
Blood ; 131(3): 328-341, 2018 01 18.
Article em En | MEDLINE | ID: mdl-29113963
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteínas Repressoras / Leucemia Mieloide / Mutação com Ganho de Função Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Animals Idioma: En Revista: Blood Ano de publicação: 2018 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteínas Repressoras / Leucemia Mieloide / Mutação com Ganho de Função Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Animals Idioma: En Revista: Blood Ano de publicação: 2018 Tipo de documento: Article