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Dihydroartemisinin ameliorates LPS-induced neuroinflammation by inhibiting the PI3K/AKT pathway.
Gao, Yuting; Cui, Miaomiao; Zhong, Sijin; Feng, Chenyao; Nwobodo, Alexander Kenechukwu; Chen, Bin; Song, Yuanjian; Wang, Yulan.
Afiliação
  • Gao Y; Medical Technology School, Xuzhou Medical University, Xuzhou, 221004, China.
  • Cui M; Department of Genetics, Xuzhou Medical University, 209 Tongshan Road, Xuzhou, Jiangsu, 221004, People's Republic of China.
  • Zhong S; Department of Clinical, Xuzhou Medical University, Xuzhou, China.
  • Feng C; Department of Clinical, Xuzhou Medical University, Xuzhou, China.
  • Nwobodo AK; Department of Genetics, Xuzhou Medical University, 209 Tongshan Road, Xuzhou, Jiangsu, 221004, People's Republic of China.
  • Chen B; Department of Rehabilitation, The Affiliated People's Hospital of Fujian University of Traditional Chinese Medicine, Fuzhou, 350004, Fujian Province, China.
  • Song Y; Department of Genetics, Xuzhou Medical University, 209 Tongshan Road, Xuzhou, Jiangsu, 221004, People's Republic of China. biosongyuanjian@126.com.
  • Wang Y; Department of Human Anatomy, Xuzhou Medical University, Xuzhou, Jiangsu, 221004, People's Republic of China. ylwang-xz@163.com.
Metab Brain Dis ; 35(4): 661-672, 2020 04.
Article em En | MEDLINE | ID: mdl-32152798
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Transdução de Sinais / Fosfatidilinositol 3-Quinases / Artemisininas / Proteínas Proto-Oncogênicas c-akt / Inflamação / Anti-Inflamatórios Limite: Animals Idioma: En Revista: Metab Brain Dis Assunto da revista: CEREBRO / METABOLISMO Ano de publicação: 2020 Tipo de documento: Article País de afiliação: China
Texto completo
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XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Transdução de Sinais / Fosfatidilinositol 3-Quinases / Artemisininas / Proteínas Proto-Oncogênicas c-akt / Inflamação / Anti-Inflamatórios Limite: Animals Idioma: En Revista: Metab Brain Dis Assunto da revista: CEREBRO / METABOLISMO Ano de publicação: 2020 Tipo de documento: Article País de afiliação: China